H
Henry Masur
Researcher at National Institutes of Health
Publications - 414
Citations - 56261
Henry Masur is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Pneumocystis carinii & Pneumonia. The author has an hindex of 105, co-authored 402 publications receiving 52273 citations. Previous affiliations of Henry Masur include Burroughs Wellcome Fund & Memorial Sloan Kettering Cancer Center.
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HIV infection and cardiovascular disease--is there really a link?
Peter Sklar,Henry Masur +1 more
TL;DR: Soon after the introduction of protease inhibitors and nonnucleoside reverse-transcriptase inhibitors for the management of human immunodeficiency virus (HIV) infection, clinicians observed unexpec...
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Induced sputum to diagnose Pneumocystis carinii pneumonia in immunosuppressed pediatric patients.
Frederick P. Ognibene,Vee J. Gill,Philip A. Pizzo,Joseph A. Kovacs,Cynthia Godwin,Anthony F. Suffredini,James H. Shelhame,Joseph E. Parrillo,Henry Masur +8 more
TL;DR: In summary abnormalities of neutrophil function develop in patients with ketosis induced by any of a variety of manipulations or disease states, and consideration should be given to discontinuing iatrogenic causes of ketosis in such patients if bacterial infections develop.
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Interferon-α Produces Significant Decreases in HIV Load
Jorge A. Tavel,Chiung Yu Huang,Jean Shen,Julia A. Metcalf,Robin L. Dewar,Akram S. Shah,M. B. Vasudevachari,Dean Follmann,Betsey Herpin,Richard T. Davey,Michael A. Polis,Joseph A. Kovacs,Henry Masur,H. Clifford Lane +13 more
TL;DR: There was no significant difference among groups in change in total CD4(+) count or in time to AIDS or death, and since treatment with IFN-alpha produces significant decreases in HIV load, additional studies of IFn-alpha as part of a combination regimen are warranted.
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Sera from patients with the acquired immunodeficiency syndrome inhibit production of interleukin-2 by normal lymphocytes.
TL;DR: Sera from patients with the acquired immunodeficiency syndrome (AIDS) suppressed phytohemagglutinin (PHA)-induced IL-2 production by normal blood mononuclear cells and acted directly on the groups of cells that produce IL-1, T cells and large granular lymphocytes; suppression occurred at an early, probably pretranslational stage.
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Guide to Major Clinical Trials of Antiretroviral Therapy in Human Immunodeficiency Virus-Infected Patients: Protease Inhibitors, Non-Nucleoside Reverse Transcriptase Inhibitors, and Nucleotide Reverse Transcriptase Inhibitors
TL;DR: The intent is to present a thorough, but not necessarily exhaustive, review of studies that have shaped the principles of antiretroviral therapy.