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Hieronymus A.M. Voorbij

Researcher at Utrecht University

Publications -  43
Citations -  1517

Hieronymus A.M. Voorbij is an academic researcher from Utrecht University. The author has contributed to research in topics: Paraoxonase & Population. The author has an hindex of 23, co-authored 43 publications receiving 1429 citations. Previous affiliations of Hieronymus A.M. Voorbij include University Medical Center Utrecht.

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Risk factors for cardiovascular disease in women with a history of pregnancy complicated by preeclampsia or intrauterine growth restriction.

TL;DR: Preeclampsia or IUGR may represent an early marker for increased risk for early cardiovascular disease and show a tendency to higher BMI, higher triglyceride concentrations, and increased insulin resistance as compared with women with a history of normal pregnancy.
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Inflammatory mediators for the diagnosis and treatment of sepsis in early infancy

TL;DR: IL-6, IL-8, and PCT are better parameters than CRP in the diagnosis and follow-up of neonatal sepsis due to coagulase-negative staphylococci (CoNS) and in the exclusion of bacterial infection among those with enteroviral infection among febrile infants presenting from home.
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Serum Ferritin Is a Risk Factor for Stroke in Postmenopausal Women

TL;DR: Higher serum ferritin concentrations in postmenopausal women are associated with an increased risk of ischemic stroke, but neither serum iron nor transferrin saturation was associated with a increased stroke risk.
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Indications that paraoxonase-1 contributes to plasma high density lipoprotein levels in familial hypercholesterolemia.

TL;DR: In this paper, the effects of PON1 levels, activity, and genetic variation on high density lipoprotein-cholesterol (HDL-C) levels, circulating oxidized LDL (OxLDL), subclinical inflammation [high-sensitive C-reactive protein (Hs-CRP), and carotid atherosclerosis were determined in 302 patients with familial hypercholesterolemia.
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PON2 gene variants are associated with clinical manifestations of cardiovascular disease in familial hypercholesterolemia patients.

TL;DR: The data indicate that the common PON2 polymorphism is associated with clinical manifestations of CVD in FH patients, and subjects with the Cys/Cys311 genotype are likely to be protected against the development of premature CVD.