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Hong Li

Researcher at Guangzhou University of Chinese Medicine

Publications -  23
Citations -  1282

Hong Li is an academic researcher from Guangzhou University of Chinese Medicine. The author has contributed to research in topics: Muscle hypertrophy & Angiotensin II. The author has an hindex of 14, co-authored 20 publications receiving 836 citations. Previous affiliations of Hong Li include Sun Yat-sen University & National Institutes of Health.

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The orphan receptor NOR1 participates in isoprenaline-induced cardiac hypertrophy by regulating PARP-1.

TL;DR: The roles of NOR1 in cardiac hypertrophy induced by isoprenaline and the underlying molecular mechanisms were investigated and its potential contribution to cardiovascular diseases remains to be assessed.
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Mitochondrial binding of α-enolase stabilizes mitochondrial membrane: its role in doxorubicin-induced cardiomyocyte apoptosis.

TL;DR: The present study indicates that mitochondria-located α-enolase has a beneficial role in stabilizing mitochondrial membrane, and in cardiomyocytes, the displacement of α- enolase from mitochondria by doxorubicin may involve in activation of the intrinsic cell death pathway.
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COX-2 is involved in ET-1-induced hypertrophy of neonatal rat cardiomyocytes: role of NFATc3.

TL;DR: In the cultured rat neonatal cardiomyocytes, ET-1 significantly upregulated the expression and activity of COX-2, which was accompanied by increase in cell surface area and BNP mRNA level, and this report suggested a potential therapeutic strategy against cardiac hypertrophy by inhibiting COx-2.
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Histone Deacetylases (HDACs) and Atherosclerosis: A Mechanistic and Pharmacological Review.

TL;DR: An updated view on the roles of HDACs (Class I, Class II, Class IV) and HDAC inhibitors in vascular dysfunction and AS is presented and the novel therapeutic targets and agents in atherosclerotic cardiovascular diseases are elaborate.
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Dkk1 exacerbates doxorubicin-induced cardiotoxicity by inhibiting the Wnt/β-catenin signaling pathway.

TL;DR: The crucial role of the Dkk1–Wnt/β-catenin signaling axis has a crucial role in Dox-induced cardiotoxicity, suggesting a potential therapeutic approach for treatment of cardiomyopathy caused by clinical use of Dox.