H
Hongjuan Ning
Researcher at Liaoning Medical University
Publications - 4
Citations - 74
Hongjuan Ning is an academic researcher from Liaoning Medical University. The author has contributed to research in topics: Autophagy & Hypoxia (medical). The author has an hindex of 2, co-authored 4 publications receiving 18 citations.
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Journal ArticleDOI
Exosomes secreted by FNDC5-BMMSCs protect myocardial infarction by anti-inflammation and macrophage polarization via NF-κB signaling pathway and Nrf2/HO-1 axis.
TL;DR: FNDC5-BMMSCs-derived exosomes play anti-inflammation effects and promote M2 macrophage polarization via NF-κB signaling pathway and Nrf2/HO-1 Axis, which may develop a promising cell-free therapy for MI.
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Irisin ameliorates high glucose‐induced cardiomyocytes injury via AMPK/mTOR signal pathway
Jingyu Deng,Ning Zhang,Feng Chen,Chao Yang,Hongjuan Ning,Chun Xiao,Ke Sun,Yongfei Liu,Ming Yang,Taohong Hu,Zheng Zhang,Wei Jiang +11 more
TL;DR: Irisin plays a significant role in antiapoptosis, anti‐inflammation, antioxidative stress in HG‐induced CMs via AMPK/mammalian target of the rapamycin signaling pathway and is reversed after the administration of Compound C, AMP‐activated protein kinase (AMPK) inhibitor.
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Inositol pyrophosphates mediated the apoptosis induced by hypoxic injury in bone marrow-derived mesenchymal stem cells by autophagy
Jingyu Deng,Chao Yang,Yong Wang,Ming Yang,Haixu Chen,Hongjuan Ning,Chengzhu Wang,Yanjun Liu,Zheng Zhang,Taohong Hu +9 more
TL;DR: It is indicated that hypoxia increased autophagy and apoptosis via IP7-mediated Akt/mTOR signaling pathway of BM-MSCs and may provide a new potential therapy target for myocardial infarction.
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β-arrestin1 inhibits hypoxic injury-induced autophagy in human pulmonary artery endothelial cells via the Akt/mTOR signaling pathway.
TL;DR: It is demonstrated, for the first time, that β-arrestin1 reduces excessive autophagy and apoptosis resistance by activating the Akt/mTOR axis in hypoxic hPAECs.