H
Hyo Ihl Chang
Researcher at Korea University
Publications - 79
Citations - 2200
Hyo Ihl Chang is an academic researcher from Korea University. The author has contributed to research in topics: Bacteriophage & Temperateness. The author has an hindex of 24, co-authored 79 publications receiving 2013 citations.
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Journal ArticleDOI
A novel electrochemically active and Fe(III)-reducing bacterium phylogenetically related to Clostridium butyricum isolated from a microbial fuel cell
Hyung Soo Park,Byung Hong Kim,Hyo Suk Kim,Hyung Joo Kim,Gwang Tae Kim,Mia Kim,In Seop Chang,Yong Keun Park,Hyo Ihl Chang +8 more
TL;DR: Cyclic voltammetry showed that Clostridium butyricum EG3 cells were electrochemically active, a novel characteristic of strict anaerobic Gram-positive bacteria, suggesting that Fe(III) ion is utilised as an electron sink.
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The natural carotenoid astaxanthin, a PPAR-α agonist and PPAR-γ antagonist, reduces hepatic lipid accumulation by rewiring the transcriptome in lipid-loaded hepatocytes.
Yaoyao Jia,Jin Young Kim,Hee Jin Jun,Sun Joong Kim,Ji Hae Lee,Minh Hien Hoang,Kwang Yeon Hwang,Soo-Jong Um,Hyo Ihl Chang,Sung Joon Lee +9 more
TL;DR: AX is a PPAR-α agonist and PPar-γ antagonist, reduces hepatic lipid accumulation by rewiring the transcriptome in lipid-loaded hepatocytes by measuring peroxisome proliferator-activated receptors (PPAR) activity.
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Protective effect of astaxanthin on naproxen-induced gastric antral ulceration in rats.
TL;DR: Results suggest that astaxanthin removes the lipid peroxides and free radicals induced by naproxen, and it may offer potential remedy of gastric ulceration.
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Effects of astaxanthin on the production of NO and the expression of COX-2 and iNOS in LPS-stimulated BV2 microglial cells.
TL;DR: The results suggest that astaxanthin, probably due to its antioxidant activity, inhibits the production of inflammatory mediators by blocking iNOS and COX-2 activation or by the suppression of iN OS and COx-2 degradation.
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Cyanidin is an agonistic ligand for peroxisome proliferator-activated receptor-alpha reducing hepatic lipid.
Yaoyao Jia,Jin Young Kim,Hee Jin Jun,Sun Joong Kim,Ji Hae Lee,Minh Hien Hoang,Hyun Sook Kim,Hyo Ihl Chang,Kwang Yeon Hwang,Soo-Jong Um,Sung Joon Lee +10 more
TL;DR: Cyanidin likely acts as a physiological PPARα agonist and potentially for PPARβ/δ and γ, and reduces hepatic lipid concentrations by rewiring the expression of genes involved in lipid metabolic pathways.