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Ian M. Colrain

Researcher at Oklahoma State University Center for Health Sciences

Publications -  176
Citations -  9013

Ian M. Colrain is an academic researcher from Oklahoma State University Center for Health Sciences. The author has contributed to research in topics: Polysomnography & Non-rapid eye movement sleep. The author has an hindex of 50, co-authored 172 publications receiving 7404 citations. Previous affiliations of Ian M. Colrain include University of Auckland & University of Tasmania.

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A review of the evidence for P2 being an independent component process: age, sleep and modality.

TL;DR: The evidence supportive of P2 being the result of independent processes is described and several features, such as its persistence from wakefulness into sleep, the general consensus that unlike most other EEG phenomena it increases with age, and the fact that it can be generated using respiratory stimuli are highlighted.
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Changes in Sleep as a Function of Adolescent Development

TL;DR: While changes in sleep across adolescence are a normal part of development, many adolescents are getting insufficient sleep and are consequently, less likely to perform well at school, more likely to develop mood-related disturbances, be obese, and are at greater risk for traffic accidents, alcohol and drug abuse.
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A validation study of Fitbit Charge 2™ compared with polysomnography in adults.

TL;DR: Fitbit Charge 2™ shows promise in detecting sleep-wake states and sleep stage composition relative to gold standard PSG, particularly in the estimation of REM sleep, but with limitations in N3 detection.
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The effects of normal aging on sleep spindle and K-complex production.

TL;DR: The age-related decrease in sleep spindles and K-complex density is consistent with previous reports and may be interpreted as an age- related alteration of thalamocortical regulatory mechanisms.
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Association Between Apolipoprotein E ∊4 and Sleep-Disordered Breathing in Adults

TL;DR: A significant portion of sleep-disordered breathing is associated with ApoE ∊4 in the general population, and these effects increased with the number ofApoE∊4 alleles carried.