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Ibolya Czegle
Researcher at Semmelweis University
Publications - 8
Citations - 232
Ibolya Czegle is an academic researcher from Semmelweis University. The author has contributed to research in topics: Endoplasmic reticulum & Glutathione reductase. The author has an hindex of 5, co-authored 7 publications receiving 218 citations.
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Uncoupled redox systems in the lumen of the endoplasmic reticulum. Pyridine nucleotides stay reduced in an oxidative environment.
Simona Piccirella,Ibolya Czegle,Beáta Lizák,Éva Margittai,Silvia Senesi,Eszter Papp,Miklós Csala,Miklós Csala,Rosella Fulceri,Peter Csermely,József Mandl,József Mandl,Angelo Benedetti,Gábor Bánhegyi,Gábor Bánhegyi,Gábor Bánhegyi +15 more
TL;DR: The results demonstrated the separate existence of two redox systems in the endoplasmic reticulum lumen, which explains the contemporary functioning of oxidative folding and of powerful reductive reactions.
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Increased circulating interleukin-17 levels in preeclampsia.
TL;DR: Serum IL-17 levels are increased in preeclampsia, which may contribute to the development of the excessive systemic inflammatory response characteristic of the maternal syndrome of the disease.
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Multiple roles of glucose-6-phosphatases in pathophysiology: state of the art and future trends.
Paola Marcolongo,Rosella Fulceri,Alessandra Gamberucci,Ibolya Czegle,Gábor Bánhegyi,Angelo Benedetti +5 more
TL;DR: Beside the well known key role in blood glucose homeostasis, the members of the G6PC family seem to play a role as sensors of intracellular glucose and of intraluminal glucose/glucose-6-phosphate in the endoplasmic reticulum.
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Translocon pores in the endoplasmic reticulum are permeable to small anions.
TL;DR: Translationally inactive, ribosome-bound translocons allow small anions to cross the endoplasmic reticulum membrane, which can contribute to the nonspecific substrate supply of enzymes with intraluminal active centers.
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G6PT-H6PDH-11βHSD1 triad in the liver and its implication in the pathomechanism of the metabolic syndrome.
TL;DR: This review summarizes the available data on the role of the hepatic triad and its role in the metabolic syndrome, by confronting experimental findings with clinical observations.