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Ik-Kyung Jang

Researcher at Harvard University

Publications -  427
Citations -  26768

Ik-Kyung Jang is an academic researcher from Harvard University. The author has contributed to research in topics: Myocardial infarction & Intravascular ultrasound. The author has an hindex of 73, co-authored 404 publications receiving 24255 citations. Previous affiliations of Ik-Kyung Jang include Katholieke Universiteit Leuven & Hospital General Universitario Gregorio Marañón.

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Comparison of heparin and bivalirudin in patients undergoing percutaneous coronary intervention without use of glycoprotein IIb/IIIa inhibitors

TL;DR: The primary objective of this study is to compare the association between bleeding and the use unfractionated heparin (UFH) versus bivalirudin during percutaneous coronary intervention (PCI).
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Determinants of ST-segment elevation myocardial infarction as clinical presentation of acute coronary syndrome

TL;DR: Antiplatelet therapy prior to the onset of ACS is associated with reduced probability of ST-segment elevation myocardial infarction (STEMI) following both plaque rupture and plaque erosion, and dual anti platelet therapy offers additional protection compared to a single antiplatelet agent in plaque erosion.
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Abstract 13076: Lipid-lowering Therapy Stabilizes the Complexity of Non-culprit Plaques in Human Coronary Artery: A Quantitative Assessment Using OCT Bright Spot Algorithm

TL;DR: In this paper, the authors reported that statin therapy increases the thickness of fibrous cap in human coronary plaques, however, the efficacy on the sta cation was not evaluated.
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SYNTAX Score and Pre- and Poststent Optical Coherence Tomography Findings in the Left Anterior Descending Coronary Artery in Patients With Stable Angina Pectoris.

TL;DR: Stent edge dissection and irregular protrusion were more frequent in patients with higher SS, indicating poor vascular response to stenting, which may explain higher cardiac event rate and target lesion revascularization in patientswith higher SS.
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Relation of Low-Density Lipoprotein Cholesterol Level to Plaque Rupture.

TL;DR: When LDL-C level is elevated, early and aggressive treatment with statin may help to prevent PR by stabilizing plaques through calcification, and the patients with naturally low cholesterol have the lowest risk of PR.