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Imre Kiss
Researcher at Politehnica University of Timișoara
Publications - 75
Citations - 841
Imre Kiss is an academic researcher from Politehnica University of Timișoara. The author has contributed to research in topics: Textile & Cast iron. The author has an hindex of 11, co-authored 69 publications receiving 682 citations. Previous affiliations of Imre Kiss include Semmelweis University & University of Szeged.
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Sharing secrets: oxytocin and trust in schizophrenia.
TL;DR: Low oxytocin levels measured after trust-related interactions significantly predicted the negative symptoms of schizophrenia but were not related to positive symptoms, depression, anxiety, and neuropsychological functions.
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Anomalous visual experiences, negative symptoms, perceptual organization and the magnocellular pathway in schizophrenia: a shared construct?
TL;DR: The results raise the possibility that multiple facets of visual information processing deficits can be explained by M pathway dysfunctions in schizophrenia, resulting in impaired attentional modulation of perceptual organization and of natural image categorization.
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Perceptual and cognitive effects of antipsychotics in first-episode schizophrenia: The potential impact of GABA concentration in the visual cortex
TL;DR: The results suggest that antipsychotics dominantly affect visual contrast sensitivity and anomalous perceptual experiences and the prominent dampening effect on low spatial frequency in the steady-pedestal test might indicate the normalization of putatively overactive magnocellular retino-geniculo-cortical pathways.
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Oxytocin response in a trust game and habituation of arousal.
Szabolcs Kéri,Imre Kiss +1 more
TL;DR: Results suggest that the habituation of autonomic arousal is closely related to oxytocin released during trust-related social interactions, and this relationship was absent in the case of neutral (trust-unrelated) oxytocIn level.
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When doors of perception open: visual contrast sensitivity in never-medicated, first-episode schizophrenia.
TL;DR: Results indicate the heightened sensitivity of magnocellular pathways in unmedicated first-episode schizophrenia, which may contribute to anomalous perceptual experiences and sensory overloading.