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Inger Gjertsson

Researcher at University of Gothenburg

Publications -  106
Citations -  1643

Inger Gjertsson is an academic researcher from University of Gothenburg. The author has contributed to research in topics: Medicine & Rheumatoid arthritis. The author has an hindex of 21, co-authored 79 publications receiving 1171 citations. Previous affiliations of Inger Gjertsson include Sahlgrenska University Hospital.

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Reactive Oxygen Species Produced by the NADPH Oxidase 2 Complex in Monocytes Protect Mice from Bacterial Infections

TL;DR: Monocyte/macrophage expression of functional NCF1 protected against spontaneous and administered bacterial infections, and MN+ mice survived after being administered Burkholderia cepacia, an opportunistic pathogen in CGD patients, whereas MN− mice died.
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Model systems: modeling human staphylococcal arthritis and sepsis in the mouse.

TL;DR: Murine models of Staphylocococus aureus-mediated arthritis and sepsis exist and are being used to gain a better understanding of the host-bacterium relationship as well to develop better methods of prevention and treatment.
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Anti-inflammatory Diet In Rheumatoid Arthritis (ADIRA)-a randomized, controlled crossover trial indicating effects on disease activity.

TL;DR: Positive effects of a proposed anti-inflammatory diet on disease activity in patients with RA are indicated and additional studies are required to determine if this diet can cause clinically relevant improvements.
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CD21 -/low B cells: A Snapshot of a Unique B Cell Subset in Health and Disease.

TL;DR: A B cell subset that is expanded in conditions characterized by chronic immune stimulation, which lacks, or expresses low, surface levels of the complement receptor 2 (CD21) and has therefore been termed CD21−/low B cells is discussed.
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CD21(-/low) B cells in human blood are memory cells.

TL;DR: It is shown that CD21–/low cells represent approximately 5% of B cells in peripheral blood from adults but are barely detectable in cord blood, after excluding transitional B cells, implying that some memory B cells require not only TLR but also BCR triggering.