J. Frank Nash

TL;DR: Data are suggestive that the long-term (7 day) DA neurotoxicity produced by the repeated administration of MA is mediated, in part, by a delayed increase in extracellular concentrations of GLU.

TL;DR: The public health benefits of sunscreens containing nano TiO(2) and/or ZnO outweigh human safety concerns for these UV filters, and the in vitro genotoxic and photogenotoxic profiles of these nano-structured metal oxides are of no consequence to human health.

TL;DR: In vivo microdialysis data are supportive of a state‐dependent, stimulatory role of 5‐HT in the regulation of dopamine release, and are consistent with the view that MDMA increases the extracellular concentration of 4,4‐methylenedioxymethamphetamine by facilitating carrier‐mediated 5‐ HT release, which can be enhanced greatly under conditions in which5‐HT synthesis is stimulated.

TL;DR: Determination of critical wavelength by means of UV substrate spectrophotometry provides a rapid, inexpensive, and reliable measure of broad-spectrum protection, which is largely independent of SPF, yet ensures long-wavelength UVA protection commensurate with SPF.

TL;DR: It is concluded that activation of 5- HT2 receptors is an important determinant of the acute increase in extracellular dopamine and, consequently, the long-term depletion of brain 5-HT produced by MDMA.