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J. H. Fuller

Researcher at University College London

Publications -  81
Citations -  8632

J. H. Fuller is an academic researcher from University College London. The author has contributed to research in topics: Diabetes mellitus & Risk factor. The author has an hindex of 45, co-authored 81 publications receiving 8218 citations. Previous affiliations of J. H. Fuller include Semmelweis University.

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Mortality and causes of death in the WHO Multinational Study of Vascular Disease in Diabetes.

TL;DR: This study confirms the importance of cardiovascular disease as the major cause of death in people with both types of diabetes and confirms the low excess mortality in the Tokyo cohort could have implications for the possible reduction of the burden of mortality associated with diabetes in other parts of the world.
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Mortality from coronary heart disease and stroke in relation to degree of glycaemia: the Whitehall study.

TL;DR: Within the glucose intolerant and diabetic groups the risk factors most strongly related to subsequent death from coronary heart disease were age and blood pressure, with less consistent relations for smoking, cholesterol concentration, and obesity.
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The role of circulating glucose and triglyceride concentrations and their interactions with other "risk factors" as determinants of arterial disease in nine diabetic population samples from the WHO multinational study.

TL;DR: In 9 of the 14 national samples of diabetic patients assembled for the WHO Multinational Study of Vascular Disease in Diabetes additional laboratory data made it possible to relate manifestations of macrovascular disease to blood glucose concentrations as well as to diabetes duration and to other potential determinants.
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Microvascular and acute complications in IDDM patients: the EURODIAB IDDM Complications Study

TL;DR: Microvascular and acute complications were clearly related to duration of diabetes and to glycaemic control, however, the relation of glycaemia control to raised albuminuria differed qualitatively from its relation to retinopathy.