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Jack L. Martin

Researcher at Bryn Mawr Hospital

Publications -  36
Citations -  3613

Jack L. Martin is an academic researcher from Bryn Mawr Hospital. The author has contributed to research in topics: Percutaneous coronary intervention & Conventional PCI. The author has an hindex of 19, co-authored 35 publications receiving 3283 citations. Previous affiliations of Jack L. Martin include University of Texas at Austin & University of Texas Health Science Center at San Antonio.

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Human Epicardial Adipose Tissue Is a Source of Inflammatory Mediators

TL;DR: Epicardial adipose tissue is a source of several inflammatory mediators in high-risk cardiac patients and plasma inflammatory biomarkers may not adequately reflect local tissue inflammation.
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Diabetes-Induced Oxidative Stress and Low-Grade Inflammation in Porcine Coronary Arteries

TL;DR: The involvement of AGEs in the development of accelerated coronary atherosclerosis in diabetes is suggested, as well as the mechanisms of abnormal formation of reactive oxygen species and the changes in inflammatory gene expression examined.
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Phase 2 Reentry as a Trigger to Initiate Ventricular Fibrillation During Early Acute Myocardial Ischemia

TL;DR: Acute regional myocardial ischemia results in markedly heterogeneous loss of Ito-mediated epicardial AP domes across the ischemic border, leading to phase 2 reentry, which can in turn produce an R-on-T extrasystole capable of initiating VF.
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Left Main Stenting in Comparison With Surgical Revascularization: 10-Year Outcomes of the (Left Main Coronary Artery Stenting) LE MANS Trial.

TL;DR: In patients with unprotected left main coronary artery stenosis with low and medium complexity of coexisting coronary artery disease, stenting offers numerically, but statistically nonsignificant, favorable long-term outcome up to 10 years in terms of safety and efficacy outcome measures, therefore, constitutes an alternative therapy for CABG.