scispace - formally typeset
J

Jaime Renart

Researcher at Spanish National Research Council

Publications -  68
Citations -  3301

Jaime Renart is an academic researcher from Spanish National Research Council. The author has contributed to research in topics: Podoplanin & Gene. The author has an hindex of 25, co-authored 68 publications receiving 3025 citations. Previous affiliations of Jaime Renart include Catalan Institution for Research and Advanced Studies & Stanford University.

Papers
More filters
Journal ArticleDOI

Transfer of proteins from gels to diazobenzyloxymethyl-paper and detection with antisera: a method for studying antibody specificity and antigen structure

TL;DR: A rapid and very sensitive method for detecting proteins as antigens after their separation in polyacrylamide/agarose composite gels, with or without sodium dodecyl sulfate.
Book ChapterDOI

[15] Detection of specific RNAs or specific fragments of DNA by fractionation in gels and transfer to diazobenzyloxymethyl paper

TL;DR: The efficiency of transfer is very high and independent of the size of a fragment as covalent linkage allows convenient multiple reuse of the transfers and also allows washings to be done repeatedly under stringent conditions, thereby helping to achieve very low backgrounds.
Journal ArticleDOI

Predictive value of nuclear factor κB activity and plasma cytokine levels in patients with sepsis

TL;DR: Results underscore the prognostic value of early measurement of NF-κB activity in patients with severe sepsis and show the interleukin-1 receptor antagonist was the only cytokine tested whose level in plasma was of value in predicting mortality by logistic regression analysis.
Journal ArticleDOI

Podoplanin in Inflammation and Cancer.

TL;DR: The diverse roles of podoplanin in inflammation and cancer are described, the protein ligands of Podoplanin identified so far are depicted, and the mechanistic basis for the involvement of pod planetin in all these processes is discussed.
Journal ArticleDOI

Interleukin-1β Enhances GABAA Receptor Cell-surface Expression by a Phosphatidylinositol 3-Kinase/Akt Pathway RELEVANCE TO SEPSIS-ASSOCIATED ENCEPHALOPATHY

TL;DR: Clinical and experimental evidence that this brain dysfunction may be related to altered neurotransmission produced by inflammatory mediators is presented and it is proposed that through this mechanism IL-1beta might alter synaptic strength at central GABAergic synapses and so contribute to the cognitive dysfunction observed in SAE.