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Jason Travis

Researcher at Translational Genomics Research Institute

Publications -  7
Citations -  325

Jason Travis is an academic researcher from Translational Genomics Research Institute. The author has contributed to research in topics: Context (language use) & Genomics. The author has an hindex of 4, co-authored 7 publications receiving 226 citations.

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NASP: an accurate, rapid method for the identification of SNPs in WGS datasets that supports flexible input and output formats.

TL;DR: This study demonstrates how NASP compares with other tools in the analysis of two real bacterial genomics datasets and one simulated dataset and demonstrates differences in results based on the choice of the reference genome and choice of inferring phylogenies from concatenated SNPs or alignments including monomorphic positions.
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A unique multidrug-resistant clonal Trichophyton population distinct from Trichophyton mentagrophytes/Trichophyton interdigitale complex causing an ongoing alarming dermatophytosis outbreak in India: Genomic insights and resistance profile.

TL;DR: This study highlights difficulties in identifying isolates from the Trichophyton mentagrophytes/interdigitale clade of the genus using currently available molecular tools and highlights the potential of this organism to rapidly spread alleles that might be driving antifungal resistance among its population.
Posted ContentDOI

The Northern Arizona SNP Pipeline (NASP): accurate, flexible, and rapid identification of SNPs in WGS datasets

TL;DR: This study demonstrates how NASP compares to other tools in the analysis of two real bacterial genomics datasets and one simulated dataset and demonstrates differences in results based on the choice of the reference genome and choice of inferring phylogenies from concatenated SNPs or alignments including monomorphic positions.
Journal ArticleDOI

Rhizopus microsporus Infections Associated with Surgical Procedures, Argentina, 2006-2014.

TL;DR: In this article, the authors performed whole-genome sequencing on samples collected during 2006-2014 and found no evidence for recombination or nonneutral mutation accumulation; these findings do not support common source or patient-to-patient transmission.