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Jean-Pierre Dedet

Researcher at Pasteur Institute

Publications -  49
Citations -  1079

Jean-Pierre Dedet is an academic researcher from Pasteur Institute. The author has contributed to research in topics: Leishmaniasis & Cutaneous leishmaniasis. The author has an hindex of 19, co-authored 49 publications receiving 1042 citations.

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Leishmania infantum and L. major in Algeria.

TL;DR: Since 1980, the development of leish maniasis in Algeria has been marked by a considerable increase in the number of cases of both visceral leishmaniasis and cutaneous leishManiasis (more than 2000 cases per year), with major foci in the north and south of the country.
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Sudan: the possible original focus of visceral leishmaniasis.

TL;DR: The phylogenetic analysis showed that the 7 Leishmania zymodemes obtained hold ancestral positions on the phylogenetic tree, supporting the hypothesis of an East African origin of visceral leishmaniasis.
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Epidemiological aspects of human cutaneous leishmaniasis in French Guiana.

TL;DR: A follow-up study of 219 patients infected with parasitologically confirmed cutaneous leishmaniasis in French Guiana was made between 1981 and 1987, finding that young male adults entering the forest for professional activities and having lesions of the classical ulcerative type appeared to be common.
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Diffuse cutaneous infection caused by a presumed monoxenous trypanosomatid in a patient infected with HIV

TL;DR: A patient infected with human immunodeficiency virus developed a diffuse cutaneous nodular syndrome and the parasite isolated from a skin nodule was studied by isoenzymatic characterization and transmission electron microscopy of both culture forms and those in the patient's skin biopsy.
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A previously unclassified trypanosomatid responsible for human cutaneous lesions in Martinique (French West Indies) is the most divergent member of the genus Leishmania ss.

TL;DR: Two cases of skin lesions similar to those caused by Leishmania parasites have been reported from Martinique and sequence of the 18S ribosomal RNA gene and partial sequences of the DNA polymerase alpha and RNA polymerase II largest subunit genes indicated that the Martinique parasites clustered with L. enriettii and were basal to all other euleishmania.