J
Jiang-Ning Zhou
Researcher at University of Science and Technology of China
Publications - 178
Citations - 9746
Jiang-Ning Zhou is an academic researcher from University of Science and Technology of China. The author has contributed to research in topics: Hypothalamus & Circadian rhythm. The author has an hindex of 49, co-authored 172 publications receiving 8631 citations. Previous affiliations of Jiang-Ning Zhou include Center for Excellence in Education & Anhui Medical University.
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A sex difference in the human brain and its relation to transsexuality.
TL;DR: This study is the first to show a female brain structure in genetically male transsexuals and supports the hypothesis that gender identity develops as a result of an interaction between the developing brain and sex hormones.
Journal Article
A sex difference in the human brain and its relation to transsexuality
TL;DR: The authors found that the volume of the central subdivision of the bed nucleus of the stria terminalis (BSTc), a brain area that is essential for sexual behaviour, is larger in men than in women.
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Male-to-Female Transsexuals Have Female Neuron Numbers in a Limbic Nucleus
Frank P.M. Kruijver,Jiang-Ning Zhou,Chris W. Pool,Michel A. Hofman,Louis J. G. Gooren,Dick F. Swaab +5 more
TL;DR: The present findings of somatostatin neuronal sex differences in the BSTc and its sex reversal in the transsexual brain clearly support the paradigm that in transsexuals sexual differentiation of the brain and genitals may go into opposite directions and point to a neurobiological basis of gender identity disorder.
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Suppression of neuroinflammation by astrocytic dopamine D2 receptors via αB-crystallin
Wei Shao,Shu-zhen Zhang,Mi Tang,Mi Tang,Xinhua Zhang,Zheng Zhou,Yanqing Yin,Qinbo Zhou,Yuan-yuan Huang,Yingjun Liu,Eric F. Wawrousek,Teng Chen,Sheng-bin Li,Ming Xu,Jiang-Ning Zhou,Gang Hu,Jiawei Zhou +16 more
TL;DR: It is shown that the astrocytic dopamine D2 receptor (DRD2) modulates innate immunity through αB-crystallin (CRYAB), which is known to suppress neuroinflammation in the central nervous system through a CRYAB-dependent mechanism.
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Decreased melatonin levels in postmortem cerebrospinal fluid in relation to aging, Alzheimer's disease, and apolipoprotein E-epsilon4/4 genotype.
TL;DR: A dramatic decrease in the CSF melatonin levels was found in old control subjects and even more so inAD patients, and whether supplementation of melatonin may indeed improve behavioral disturbances in AD patients should be investigated.