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Jie Zhang

Researcher at China Agricultural University

Publications -  14
Citations -  836

Jie Zhang is an academic researcher from China Agricultural University. The author has contributed to research in topics: Gut flora & Immune system. The author has an hindex of 10, co-authored 14 publications receiving 493 citations. Previous affiliations of Jie Zhang include United States Department of Agriculture.

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Nutrients Mediate Intestinal Bacteria–Mucosal Immune Crosstalk

TL;DR: This review focuses on the recent discoveries about intestinal bacteria–immune crosstalk and nutritional regulation on their interplay, with an aim to provide novel insights that can aid in understanding their interactions.
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Autophagy: The Last Defense against Cellular Nutritional Stress.

TL;DR: The process of nutritional stress-induced autophagy and its important physiologic and/or pathologic roles in cell metabolism and diseases are highlighted, and light is shed on the research into the mechanism and clinical applications of Autophagy induced by nutritional stresses.
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Melatonin mediates mucosal immune cells, microbial metabolism, and rhythm crosstalk: A therapeutic target to reduce intestinal inflammation.

TL;DR: Unclear mechanisms and undiscovered actions of melatonin in gut‐microbiome‐immune axis are revealed and provide new insight for the outlook ofmelatonin as a potential therapeutic target in the treatment and management of intestinal diseases.
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Dietary Fiber Increases Butyrate-Producing Bacteria and Improves the Growth Performance of Weaned Piglets

TL;DR: Dietary CB or WB may enhance the growth performance of weaned piglets via altering gut microbiota and improving butyrate production, which shed light on the mechanism of dietary fiber in improving gut health.
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Moderate Dietary Protein Restriction Optimized Gut Microbiota and Mucosal Barrier in Growing Pig Model.

TL;DR: Moderate protein restriction (15% CP) can optimize the ileal microbiota structure via strengthening beneficial microbial populations and suppressing harmful bacterial growth and altering the function of ilesal tight junction proteins as well as epithelial cell proliferation.