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Jing Qi

Researcher at Chinese PLA General Hospital

Publications -  7
Citations -  221

Jing Qi is an academic researcher from Chinese PLA General Hospital. The author has contributed to research in topics: Sleep deprivation & Anterior cingulate cortex. The author has an hindex of 2, co-authored 6 publications receiving 101 citations. Previous affiliations of Jing Qi include Nankai University.

Papers
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The evaluation of sleep disturbances for Chinese frontline medical workers under the outbreak of COVID-19.

TL;DR: FMW have higher prevalence of sleep disturbances and worse sleep quality than non-FMW, and further interventions should be administrated for FMW, aiming to maintain their healthy condition and guarantee their professional performance in the battle against COVID-19.
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Altered insula-prefrontal functional connectivity correlates to decreased vigilant attention after total sleep deprivation.

TL;DR: In this article, the authors explored the alterations in insular functional connectivity and its association with vigilant attention performance following total sleep deprivation (TSD) and found that the insula is a key hub of the salience network that mediates shifting attention between endogenous and exogenous states.
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Disrupted Small-world Networks are Associated with Decreased Vigilant Attention after Total Sleep Deprivation.

TL;DR: In this paper, the topological properties of brain networks derived from resting-state functional magnetic resonance imaging of 26 healthy men in rested wakefulness (RW) state and after 36h of total sleep deprivation (TSD) were evaluated based on graph theory analysis.
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Altered functional connectivity between the nucleus basalis of Meynert and anterior cingulate cortex is associated with declined attentional performance after total sleep deprivation.

TL;DR: The results suggest that the disrupted NBM-related cholinergic circuit highlights an important role in attentional performance after TSD, and the enhanced NBM functional connectivity with the anterior cingulate cortex may act as neural signatures for attentional deficits induced by sleep deprivation.