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Jinghua Peng

Researcher at Shanghai University

Publications -  52
Citations -  1336

Jinghua Peng is an academic researcher from Shanghai University. The author has contributed to research in topics: Fatty liver & Liver injury. The author has an hindex of 15, co-authored 48 publications receiving 1009 citations.

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Roles of Chlorogenic Acid on Regulating Glucose and Lipids Metabolism: A Review

TL;DR: This review addresses current studies investigating the roles of CGA in glucose and lipid metabolism and indicates that CGA exerts many biological properties, including antibacterial, antioxidant, and anticarcinogenic activities.
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Gut Microbial Dysbiosis Is Associated with Altered Hepatic Functions and Serum Metabolites in Chronic Hepatitis B Patients.

TL;DR: In this article, the compositional and functional changes in the gut microbiota in early stage chronic hepatitis B (CHB) patients suggest the potential contributions of gut microbiota to the progression of CHB, and thus provide new insight into gut microbiota-targeted interventions to improve the prognosis of this disease.
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Structural changes of gut microbiota in a rat non-alcoholic fatty liver disease model treated with a Chinese herbal formula.

TL;DR: The results revealed that the bacterial profiles of HFD-induced rats could be modulated by theCHF, and differences in microbiota composition provided a basis for further understanding the pharmacological mechanism of the CHF.
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Anti-fibrotic effect of Cordyceps sinensis polysaccharide: Inhibiting HSC activation, TGF-β1/Smad signalling, MMPs and TIMPs.

TL;DR: It is indicated that the effects of CS-PS anti-liver fibrosis are probably associated with the inhibition on HSC activation, TGF-β1/Smads signalling pathway, as well as MMP2, MMP9 activity and TIMP2 expression.
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Chinese medicine CGA formula ameliorates DMN-induced liver fibrosis in rats via inhibiting MMP2/9, TIMP1/2 and the TGF-β/Smad signaling pathways.

TL;DR: Chinese medicine CGA formula ameliorates DMN-induced liver fibrosis in rats, and this effect was likely associated with the down-regulation of MMP2/9 activities, TIMP1/2 protein expression and the TGF-β1/Smad signaling pathways in the liver.