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Joanna H. Queen

Researcher at University of Texas at Austin

Publications -  6
Citations -  453

Joanna H. Queen is an academic researcher from University of Texas at Austin. The author has contributed to research in topics: Temperature-dependent sex determination & Turtle (robot). The author has an hindex of 5, co-authored 6 publications receiving 422 citations.

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Expression of Sox9, Mis, and Dmrt1 in the gonad of a species with temperature‐dependent sex determination

TL;DR: It is found the first evidence that Sox9 expression is preferentially organized in the testis early in the temperature‐sensitive period in a species with temperature‐dependent sex determination (Trachemys scripta).
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Cloning and expression of R-Spondin1 in different vertebrates suggests a conserved role in ovarian development.

TL;DR: Cl cloning and comparative expression analysis of R-SPONDIN1 orthologues in the mouse, chicken and red-eared slider turtle, three species with different sex-determining mechanisms, indicate that RSPO1 is up-regulated in the embryonic gonads of female vertebrates with differentsex-d determining mechanisms and suggest that this gene is an ancient, conserved part of the vertebrate ovary-d determining pathway.
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Response of candidate sex-determining genes to changes in temperature reveals their involvement in the molecular network underlying temperature-dependent sex determination.

TL;DR: In this article, the response of six candidate sex-determining genes to sex-reversing temperature shifts in a species with temperature-dependent sex determination was examined, including FoxL2, Wnt4, Dmrt1, and Mis by temperature.

Response of candidate sex-determining genes to changes in temperature reveals their involvement in the molecular network underlying temperature-dependent sex determination Running title: Temperature-responsive genes involved in gonadogenesis

TL;DR: Examining the role of FoxL2, Wnt4, Dmrt1, and Mis by temperature can begin to elucidate elements of conservation and divergence between sex-determining mechanisms and reveal that at the level of gene expression, response to temperature is comparatively later in gonadogenesis.
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Spinal cord injury causes sustained disruption of the blood-testis barrier in the rat.

TL;DR: SCI is followed by compromised BTB integrity by as early as 72 hours post-injury in rats and is accompanied by a substantial immune response within the testis, which remains compromised and testis immune cell infiltration persists for months after the initial injury.