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John C. Quindry

Researcher at University of Montana

Publications -  135
Citations -  4922

John C. Quindry is an academic researcher from University of Montana. The author has contributed to research in topics: Cardioprotection & Oxidative stress. The author has an hindex of 36, co-authored 124 publications receiving 4420 citations. Previous affiliations of John C. Quindry include Providence Regional Medical Center Everett & University of Florida.

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Exercise does not increase cyclooxygenase-2 myocardial levels in young or senescent hearts

TL;DR: Investigation of whether aerobic exercise elevates myocardial COX-2 protein content or enzyme activity in young and senescent male Fisher 344 rats assigned to sedentary orCardioprotective endurance exercise treatments suggests that mechanisms responsible for cardioprotection differ between IPC and aerobic exercise.
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Muscle-fiber type and blood oxidative stress after eccentric exercise

TL;DR: There are correlations between Type II muscle-fiber type and postexercise PC, and further study is needed to understand the mechanisms responsible for the observed fast-twitch muscle- fiber oxidative-stress relationship.
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Evaluation of arrhythmia scoring systems and exercise-induced cardioprotection.

TL;DR: Evaluating seven ECG scoring systems in the assessment of ventricular arrhythmia severity after IR in male Sprague-Dawley rats confirmed that exercise protects against IR-induced arrhythmias, and care must be taken when selecting an arrHythmia scoring system for ECG evaluation.
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Involvement of the δ‐opioid receptor in exercise‐induced cardioprotection

TL;DR: In exercised hearts, the δ‐opioid receptor appears to trigger cardioprotection against ischaemia–reperfusion‐induced tissue necrosis but not apoptosis.
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Autophagic dysfunction and autophagosome escape in the mdx mus musculus model of Duchenne muscular dystrophy.

TL;DR: The purpose of this investigation was to determine the extent to which basal autophagy changes with disease progression, and hypothesized thatautophagy impairment would increase with advanced disease.