J
John W. Finnie
Researcher at University of Adelaide
Publications - 141
Citations - 3577
John W. Finnie is an academic researcher from University of Adelaide. The author has contributed to research in topics: Head injury & Clostridium perfringens. The author has an hindex of 28, co-authored 135 publications receiving 3076 citations. Previous affiliations of John W. Finnie include South Australia Pathology.
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Journal ArticleDOI
Lymphomas in Eμ-Pim1 Transgenic Mice Exposed to Pulsed 900 MHz Electromagnetic Fields
TL;DR: Long-term intermittent exposure to RF fields can enhance the probability that mice carrying a lymphomagenic oncogene will develop lymphomas, and it is suggested that such genetically cancer-prone mice provide an experimental system for more detailed assessment of dose-response relationships for risk of cancer after RF-field exposure.
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Electrophysiological, electroanatomical, and structural remodeling of the atria as consequences of sustained obesity
Rajiv Mahajan,Dennis H. Lau,Anthony G. Brooks,Nicholas J. Shipp,Jim Manavis,John P. M. Wood,John W. Finnie,Chrishan S. Samuel,Simon G. Royce,Darragh Twomey,Shivshanker Thanigaimani,Jonathan M. Kalman,Prashanthan Sanders,Prashanthan Sanders +13 more
TL;DR: Sustained obesity results in global biatrial endocardial remodeling characterized by LA enlargement, conduction abnormalities, fractionated electrograms, increased profibrotic TGF-β1 expression, interstitial atrial fibrosis, and increased propensity for AF.
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Long-Term Exposure of Eμ-Pim1 Transgenic Mice to 898.4 MHz Microwaves does not Increase Lymphoma Incidence
TL;DR: Findings showed that long-term exposures of lymphoma-prone mice to 898.4 MHz GSM radiofrequency (RF) radiation at SARs of 0.25, 1.0, 2.0 and 4.0 W/kg had no significant effects when compared to sham-irradiated animals.
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Neuroinflammation: beneficial and detrimental effects after traumatic brain injury.
TL;DR: This review highlights the primary and secondary lesions constituting craniocerebral trauma and the main elements of neuroinflammation, one of the most important secondary events evolving after the initial traumatic insult.
Journal ArticleDOI
Traumatic brain injury.
John W. Finnie,P. C. Blumbergs +1 more
TL;DR: How different types of animal models are useful for the study of neuropathologic processes in traumatic, blunt, nonmissile head injury is discussed.