J
José R. Criado
Researcher at Scripps Research Institute
Publications - 52
Citations - 3738
José R. Criado is an academic researcher from Scripps Research Institute. The author has contributed to research in topics: Hippocampal formation & Dentate gyrus. The author has an hindex of 25, co-authored 51 publications receiving 3540 citations. Previous affiliations of José R. Criado include University of Texas Health Science Center at Houston.
Papers
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Journal ArticleDOI
Neuropsychology and neuropharmacology of P3a and P3b.
John Polich,José R. Criado +1 more
TL;DR: The findings suggest that the circuit underlying ERP generation is influenced in a different ways for acute intake and varies between chronic use levels across drugs.
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Hypocretin-1 modulates rapid eye movement sleep through activation of locus coeruleus neurons.
Patrice Bourgin,Salvador Huitron-Resendiz,Avron D. Spier,Véronique Fabre,Beatriz Morte,José R. Criado,J. Gregor Sutcliffe,Steven J. Henriksen,Luis de Lecea +8 more
TL;DR: It is proposed that hcrt receptor 1 in the LC is a key target for REM sleep regulation and might be involved in the pathophysiological mechanisms of narcolepsy.
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A cortical neuropeptide with neuronal depressant and sleep-modulating properties
Luis de Lecea,José R. Criado,Oscar Prospéro-García,Kaare M. Gautvik,Paul Schweitzer,Patria E. Danielson,Charles L. M. Dunlop,George R. Siggins,Steven J. Henriksen,J. Gregor Sutcliffe +9 more
TL;DR: Administration of cortistatin depresses neuronal electrical activity but, unlike somatostatin, induces low-frequency waves in the cerebral cortex and antagonizes the effects of acetylcholine on hippocampal and cortical measures of excitability, which suggests a mechanism for cortical synchronization related to sleep.
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Cross-linking cellular prion protein triggers neuronal apoptosis in vivo.
Laura Solforosi,José R. Criado,Dorian B. McGavern,Sebastian A. Wirz,Manuel Sanchez-Alavez,Shuei Sugama,Lorraine A. DeGiorgio,Bruce T. Volpe,Erika Wiseman,Gil C. Abalos,Eliezer Masliah,Donald H. Gilden,Michael B. A. Oldstone,Bruno Conti,R. Anthony Williamson +14 more
TL;DR: It is suggested that PrPC functions in the control of neuronal survival and provides a model to explore whether cross-linking of PrPC by oligomeric PrPSc can promote neuronal loss during prion infection.
Journal ArticleDOI
Mice devoid of prion protein have cognitive deficits that are rescued by reconstitution of PrP in neurons.
José R. Criado,Manuel Sanchez-Alavez,Bruno Conti,Jeannie L. Giacchino,Derek N. Wills,Steven J. Henriksen,Richard E. Race,Jean Manson,Bruce Chesebro,Michael B. A. Oldstone +9 more
TL;DR: Mice with a selective deletion of PrP(C) exhibited deficits in hippocampal-dependent spatial learning, but non-spatial learning remained intact, and deficits were seen in mPrP-/- mice with a homogeneous 129/Ola background and in m prisoners in the mixed mix indicating that these abnormalities were unlikely due to variability of background genes or alteration of the nearby Prnd (doppel) gene.