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Juan Carlos Gomora

Researcher at National Autonomous University of Mexico

Publications -  36
Citations -  1132

Juan Carlos Gomora is an academic researcher from National Autonomous University of Mexico. The author has contributed to research in topics: Voltage-dependent calcium channel & Patch clamp. The author has an hindex of 16, co-authored 32 publications receiving 1021 citations. Previous affiliations of Juan Carlos Gomora include University of Virginia.

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Block of cloned human T-type calcium channels by succinimide antiepileptic drugs.

Juan Carlos Gomora, +3 more
- 01 Nov 2001 - 
TL;DR: It is concluded that succinimide antiepileptic drugs are capable of blocking human T-type channels at therapeutically relevant concentrations.
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Functional expression of voltage-gated sodium channels in primary cultures of human cervical cancer.

Daniel Diaz, +8 more
- 01 Feb 2007 - 
TL;DR: The results show for the first time the functional expression of VGSC in primary cultures from human CaC, and suggest that these channels might be considered as potential molecular markers for this type of cancer.
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Molecular Pharmacology of T-type Ca2+ Channels

Tiffany N. Heady, +3 more
- 01 Apr 2001 - 
TL;DR: This review summarizes a variety of compounds that exhibit varying degrees of blocking activity towards T-type Ca2+ channels as an aid for researchers in need of antagonists to study the biophysical and pathological nature of T- type channels, as a starting point for those attempting to develop potent and selective antagonists of the channel.
Journal ArticleDOI

Cloning and expression of the human t-type channel cav3.3: insights into prepulse facilitation

Juan Carlos Gomora, +4 more
- 01 Jul 2002 - 
TL;DR: The results suggest that sequences in the distal portion of Ca(v)3.3 play a role in channel expression, similar to high voltage-activated Ca(2+) channels in that depolarizing prepulses can regulate their activity, and their carboxy termini play arole in modulating channel activity.
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Overexpression of NaV1.6 channels is associated with the invasion capacity of human cervical cancer

Everardo Hernández-Plata, +7 more
- 01 May 2012 - 
TL;DR: It is concluded that NaV1.6 is upregulated in CaC and could serve as a novel molecular marker for the metastatic behavior of this carcinoma.