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Justin T. Saunders

Researcher at Baylor College of Medicine

Publications -  9
Citations -  1060

Justin T. Saunders is an academic researcher from Baylor College of Medicine. The author has contributed to research in topics: Heart failure & Population. The author has an hindex of 5, co-authored 7 publications receiving 978 citations. Previous affiliations of Justin T. Saunders include University of Texas Southwestern Medical Center & University of Michigan.

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Genetic targeting of green fluorescent protein to gonadotropin-releasing hormone neurons: Characterization of whole-cell electrophysiological properties and morphology

TL;DR: Direct intracellular morphological assessment of GnRH dendritic morphology revealed GnRH neurons have slightly more extensive dendrites than previously reported, indicating that voltage-dependent sodium channels are involved in generating action potentials in these cells.
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Sources of Variability in Measurements of Cardiac Troponin T in a Community-Based Sample: The Atherosclerosis Risk in Communities Study

TL;DR: The observed high laboratory and intraindividual (biological) reliability of cardiac troponin T support its use for population-based research, and in clinical settings that rely on classification and serial measurements.
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Cardiomyopathy in type 2 diabetes: update on pathophysiological mechanisms.

TL;DR: In this paper, several metabolic perturbations associated with type 2 diabetes mellitus have been implicated as contributors to the heart failure risk, including alterations of cardiomyocyte metabolic substrate switching between free fatty acid (FFA) and glucose metabolism; increased FFA exposure and cellular accumulation; and alterations in peroxisome proliferator-activated receptor-(PPAR-) alpha activity, among others.
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Variability and persistence of aspirin response in lower extremity peripheral arterial disease patients.

TL;DR: In this paper, the prevalence of poor response to aspirin (ASA) therapy over 12-month follow-up in patients with lower extremity peripheral arterial disease (PAD), and to compare the classification agreement among different ASA response assays was determined.