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K. G. M. M. Alberti

Researcher at Newcastle University

Publications -  106
Citations -  19091

K. G. M. M. Alberti is an academic researcher from Newcastle University. The author has contributed to research in topics: Insulin & Diabetes mellitus. The author has an hindex of 35, co-authored 106 publications receiving 18093 citations. Previous affiliations of K. G. M. M. Alberti include University of Benin.

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Assessing the Impact of Blood Sample Type on the Estimated Prevalence of Impaired Glucose Tolerance and Diabetes Mellitus in Epidemiological Surveys

TL;DR: Diagnostic criteria based on venous whole blood glucose and venous plasma glucose measurements are robust and give broadly comparable population prevalence of diabetes mellitus and impaired glucose tolerance irrespective of blood sample choice, it is concluded.
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Simple filter paper method for home monitoring of blood glucose, lactate, and 3-hydroxybutyrate.

TL;DR: A technique is described whereby glucose, lactate, and 3-hydroxybutyrate can be measured in capillary blood samples dried on boric acid impregnated filter paper that could prove useful in the broader metabolic assessment of the diabetic state.
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Long-term effects of a sustained-release preparation of acipimox on dyslipidemia and glucose metabolism in non—insulin-dependent diabetes mellitus☆

TL;DR: Despite the lack of change in fasting plasma NEFA levels, acipimox caused a modest beneficial improvement in overall glycemic control and plasma lipids in NIDDM patients and could be a useful agent in the treatment of dyslipidemic NID DM patients.
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Hexokinase isoenzymes in normal and cirrhotic human liver: suppression of glucokinase in cirrhosis

TL;DR: Glucokinase is dramatically suppressed in liver disease, which although partly compensated for by the increase in hexokinase I (and II), accounts in part for the well-known glucose intolerance of liver cirrhosis.
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Role of plasma non-esterified fatty acids during and after exercise.

TL;DR: Although acipimox abolished the rise in the plasma concentration of non-esterified fatty acids during exercise, there was only a 50% decrease in the rate of lipid oxidation, which suggests that an alternative source ofnon-esterification fatty acids makes an important contribution to the supply of lipid for oxidation during exercise.