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Karen J. Halliday

Researcher at University of Edinburgh

Publications -  85
Citations -  6917

Karen J. Halliday is an academic researcher from University of Edinburgh. The author has contributed to research in topics: Phytochrome & Arabidopsis. The author has an hindex of 44, co-authored 82 publications receiving 5982 citations. Previous affiliations of Karen J. Halliday include University of Leicester & University of California, Berkeley.

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The clock gene circuit in Arabidopsis includes a repressilator with additional feedback loops

TL;DR: The model provides a new conceptual framework for the plant clock that includes a three‐component repressilator circuit in its complex structure, and removes the necessity for the unknown component X (or TOC1mod) from previous clock models.
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Molecular and genetic control of plant thermomorphogenesis

TL;DR: How the emerging knowledge in Arabidopsis may be transferred to relevant crop systems is discussed, as this knowledge will be key to rational breeding for thermo-tolerant crop varieties.
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Cold and light control seed germination through the bHLH transcription factor SPATULA

TL;DR: The bHLH transcription factor SPATULA is a light-stable repressor of seed germination and mediates the germination response to temperature and SPT is required in dormant seeds for maintaining the repression of GA3ox transcription.
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The HY5-PIF regulatory module coordinates light and temperature control of photosynthetic gene transcription.

TL;DR: It is demonstrated that by directly targeting a common promoter cis-element (G-box), HY5 and PIFs form a dynamic activation-suppression transcriptional module responsive to light and temperature cues that provides a simple, direct mechanism through which environmental change can redirect transcriptional control of genes required for photosynthesis and photoprotection.
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Phytochrome control of flowering is temperature sensitive and correlates with expression of the floral integrator FT

TL;DR: In this paper, it was shown that the early flowering phenotype of phyB is temperature-dependent and that this temperature-sensitive flowering response defines a pathway that appears to be independent of the autonomous-FLC pathway.