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Kevin P. Davy

Researcher at Virginia Tech

Publications -  108
Citations -  5746

Kevin P. Davy is an academic researcher from Virginia Tech. The author has contributed to research in topics: Blood pressure & Population. The author has an hindex of 40, co-authored 103 publications receiving 5125 citations. Previous affiliations of Kevin P. Davy include University of Colorado Boulder & Colorado State University.

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Sympathetic neural activation in visceral obesity

TL;DR: The observations are consistent with the idea that abdominal visceral fat is an important adipose tissue depot linking obesity with sympathetic neural activation in humans and may have important implications for understanding the increased risk of developing cardiovascular diseases in individuals with visceral obesity.
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Obesity and hypertension: two epidemics or one?

TL;DR: The present review will provide an overview of the understanding of the etiology, pathophysiology, and treatment of obesity hypertension in humans, with a focus on the state of knowledge in humans.
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Greater rate of decline in maximal aerobic capacity with age in physically active vs. sedentary healthy women

TL;DR: The present cross-sectional findings provide additional evidence that the absolute, but not the relative, rate of decline in maximal aerobic capacity with age may be greater in highly physically active women compared with their sedentary healthy peers.
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Water consumption increases weight loss during a hypocaloric diet intervention in middle-aged and older adults.

TL;DR: When combined with a hypocaloric diet, consuming 500 ml water prior to each main meal leads to greater weight loss than a hypocoric diet alone in middle‐aged and older adults.
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Blood pressure reductions with exercise and sodium restriction in postmenopausal women with elevated systolic pressure: role of arterial stiffness.

TL;DR: Moderate SR lowers SBP and PP in postmenopausal women with elevated baseline levels more than does daily walking, and the greater blood pressure reductions with SR may be mediated in part by a decrease in the stiffness of the large elastic arteries.