K
Khalid A. Al-Ghamdi
Researcher at King Abdulaziz University
Publications - 62
Citations - 991
Khalid A. Al-Ghamdi is an academic researcher from King Abdulaziz University. The author has contributed to research in topics: Incremental sheet forming & Formability. The author has an hindex of 17, co-authored 61 publications receiving 716 citations. Previous affiliations of Khalid A. Al-Ghamdi include University of Birmingham.
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Journal ArticleDOI
Threshold tool-radius condition maximizing the formability in SPIF considering a variety of materials: Experimental and FE investigations
TL;DR: In this article, a new level of understanding on the influence of using small tool radii on the formability (θmax) is identified for single point incremental forming (SPIF), where the relative value of tool radius and blank thickness was varied over a range (from 1.1 to 3.9).
Journal ArticleDOI
Comparison of microarray expression profiles between follicular variant of papillary thyroid carcinomas and follicular adenomas of the thyroid
Hans-Juergen Schulten,Zuhoor Al-Mansouri,Ibtisam Baghallab,Nadia Bagatian,Ohoud Subhi,Sajjad Karim,Hosam Al-Aradati,Abdulmonem Al-Mutawa,Adel Johary,Abdulrahman A. Meccawy,Khalid A. Al-Ghamdi,Osman Abdel Al-Hamour,Mohammad H. Al-Qahtani,Jaudah Al-Maghrabi +13 more
TL;DR: A set of differentially expressed genes has been identified that contains valuable candidate genes to differentiate both histopathologically related tumor types on the molecular level and is one of the first studies using whole-transcript expression arrays to compare expression profiles between FVPTCs and FAs.
Journal Article
Comprehensive survey of HRAS, KRAS, and NRAS mutations in proliferative thyroid lesions from an ethnically diverse population.
Hans-Juergen Schulten,Sherine Salama,Alaa Al-Ahmadi,Zuhoor Al-Mansouri,Zeenat Mirza,Khalid A. Al-Ghamdi,Osman Abdel Al-Hamour,Etimad A. Huwait,Mamdooh Gari,Mohammad H. Al-Qahtani,Jaudah Al-Maghrabi +10 more
TL;DR: It is suggested that FAs with a RAS mutation may constitute precursor lesions for TC with follicular histology, as well as the putative NRAS hotspot region encompassing codon 97, which was sequenced in 36 thyroid lesions.
Journal Article
Mutational Screening of RET, HRAS, KRAS, NRAS, BRAF, AKT1, and CTNNB1 in Medullary Thyroid Carcinoma
Hans-Juergen Schulten,Jaudah Al-Maghrabi,Khalid A. Al-Ghamdi,Sherine Salama,Saad M. Almuhayawi,Adeel G. Chaudhary,Osman A. Hamour,Adel M. Abuzenadah,Mamdooh Gari,Mohammed H. Al-Qahtani +9 more
TL;DR: This study supports the clinical relevance of screening MTC patients for RET mutations and indicates that mutations in BRAF, AKT1, and CTNNB1 are likely not to play a role in MTC.
Journal ArticleDOI
BRAF mutations in thyroid tumors from an ethnically diverse group
Hans-Juergen Schulten,Sherine Salama,Zuhoor Al-Mansouri,Reem Alotibi,Khalid A. Al-Ghamdi,Osman Abdel Al-Hamour,Hassan Sayadi,Hosam Al-Aradati,Adel Al-Johari,Etimad A. Huwait,Mamdooh Gari,Mohammed H. Al-Qahtani,Jaudah Al-Maghrabi +12 more
TL;DR: Older age is manifold associated with unfavorable tumor markers in patients with thyroid carcinoma and the K601E identified in a PTC, FVPTC, and FTC seems to be more distributed among different histological types of TC than previously thought.