K
Khiem Tran
Researcher at University of California, Davis
Publications - 21
Citations - 3483
Khiem Tran is an academic researcher from University of California, Davis. The author has contributed to research in topics: Atopic dermatitis & Transcription factor II B. The author has an hindex of 13, co-authored 19 publications receiving 2359 citations. Previous affiliations of Khiem Tran include University of Illinois at Urbana–Champaign & University of Illinois at Chicago.
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Journal ArticleDOI
Reactive Oxygen Species in Inflammation and Tissue Injury
TL;DR: The current review compiles the past and current research in the area of inflammation with particular emphasis on oxidative stress-mediated signaling mechanisms that are involved in inflammation and tissue injury.
Journal ArticleDOI
Endothelial β-Catenin Signaling Is Required for Maintaining Adult Blood–Brain Barrier Integrity and Central Nervous System Homeostasis
Khiem Tran,Xianming Zhang,Dan Predescu,Xiaojia Huang,Roberto F. Machado,Joachim R. Göthert,Asrar B. Malik,Tibor Valyi-Nagy,You Yang Zhao +8 more
TL;DR: It is shown that endothelial &bgr;-catenin signaling is essential for maintaining BBB integrity and central nervous system homeostasis in adult mice, and the results suggest that BBB dysfunction secondary to defective &b gr;- catenin transcription activity is a key pathogenic factor in hemorrhagic stroke, seizure activity, andcentral nervous system inflammation.
Journal ArticleDOI
Large-Scale Transposon Mutagenesis of Photobacterium profundum SS9 Reveals New Genetic Loci Important for Growth at Low Temperature and High Pressure
Federico M. Lauro,Khiem Tran,Alessandro Vezzi,Nicola Vitulo,Giorgio Valle,Douglas H. Bartlett +5 more
TL;DR: These analyses of genes conditionally required for low-temperature or high-pressure growth in a deep-sea microorganism were the first global analyses of signal transduction mechanisms in adaptation to either physical parameter.
Journal ArticleDOI
Epithelial barrier dysfunctions in atopic dermatitis: a skin-gut-lung model linking microbiome alteration and immune dysregulation.
TL;DR: The mechanism by which atopic dermatitis progresses towards gastrointestinal or airway disease remains to be elucidated.
Journal ArticleDOI
Metastasis of cholangiocarcinoma is promoted by extended high-mannose glycans.
Dayoung Park,Chatchai Phoomak,Gege Xu,Laura P. Olney,Khiem Tran,Simon S. Park,Nathan E. Haigh,Guillaume Luxardi,Worachart Lert-itthiporn,Michiko Shimoda,Qiongyu Li,Nobuyuki Matoba,Fernando A. Fierro,Sopit Wongkham,Emanual Michael Maverakis,Carlito B. Lebrilla +15 more
TL;DR: It is shown that extended high-mannose glycans are more abundantly expressed in metastatic cholangiocarcinoma than in the parental tumor cells from which they were derived, providing support that α-1,2-mannosylated N-glycans present on cancer cell membrane proteins may serve as therapeutic targets for preventing metastasis.