Knut Dietzmann

TL;DR: The data strongly suggest that a specific decrease in the amount of all mitochondrial inner membrane enzymes, most probably as result of Ca2+ overload of muscle fibers, is the reason for the bioenergetic deficits in dystrophin-deficient skeletal muscle.

TL;DR: The frequent overexpression of the sst2A receptor may explain the high tracer uptake often observed in meningioma patients during somatostatin receptor scintigraphy and this simple immunohistochemical method could prove useful in identifying those cases of recurrent disease that may possibly respond to therapy with sst 2-selective agonists.

TL;DR: The data suggest that the same mechanisms that generate inherited mtDNA polymorphisms are strongly enhanced in gliomas and produce somatic mutations.

TL;DR: Both MAPK and PI3K/Akt pathways are activated at different levels in benign and malignant meningiomas, whereas MAPK activation is involved in both proliferation and apoptosis of malignant males.

TL;DR: PLK-mRNA, found in all tumour specimens investigated emphasizes the close correlation to proliferation and growth and there was a significant correlation between the PLK expression and the nuclear immunoreactivity of MIB-1.