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Kornélia Kis

Researcher at University of Szeged

Publications -  9
Citations -  614

Kornélia Kis is an academic researcher from University of Szeged. The author has contributed to research in topics: Keratinocyte & HaCaT. The author has an hindex of 8, co-authored 9 publications receiving 570 citations.

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Propionibacterium acnes and lipopolysaccharide induce the expression of antimicrobial peptides and proinflammatory cytokines/chemokines in human sebocytes

TL;DR: Evidence is provided that sebocytes are capable of producing proinflammatory cytokines/chemokines and antimicrobial peptides, which may have a role in acne pathogenesis, and that different strains of P. acnes vary in their capacity to stimulate an inflammatory response within the pilosebaceous follicle.
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Microbial compounds induce the expression of pro-inflammatory cytokines, chemokines and human β-defensin-2 in vaginal epithelial cells

TL;DR: Evidence that microbial compounds induce the production of pro-inflammatory cytokines, chemokines and antimicrobial peptides in vaginal epithelial cells is provided.
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Interleukin-1A +4845(G>T) polymorphism is a factor predisposing to acne vulgaris

TL;DR: A positive association was found between the minor T allele of the IL1A +4845(G>T) single nucleotide polymorphism (SNP) and acne, whereas no association was finding with respect to any alleles of the variable number of tandem repeats (VNTR) polymorphism of theIL1RN gene.
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Differential Expression of D-Type Cyclins in HaCaT Keratinocytes and in Psoriasis

TL;DR: The data suggest a possible role for D-type cyclins in the excessive basal-cell proliferation and perturbed keratinocyte differentiation in the psoriatic epidermis.
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Endogenous Phospholipid Metabolite Containing Topical Product Inhibits Ultraviolet Light-Induced Inflammation and DNA Damage in Human Skin

TL;DR: Physiogel AI cream significantly inhibited the development of UV light-induced erythema and thymine dimer formation in normal human skin, but did not alter the number of Ki67+ proliferating keratinocytes and the expression of p53 and ICAM-1.