K
Kurt A. Jellinger
Researcher at University of Vienna
Publications - 27
Citations - 2307
Kurt A. Jellinger is an academic researcher from University of Vienna. The author has contributed to research in topics: Dementia & Vascular dementia. The author has an hindex of 21, co-authored 27 publications receiving 2204 citations.
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Journal ArticleDOI
Neuropathological evaluation of mixed dementia
TL;DR: This work proposed the combination of autopsy-proven AD with multiple vascular or ischemic lesions with about 30-50 ml of infarcted/damaged brain tissue to suggest synergistic relations between these disorders.
Journal ArticleDOI
Prevalence of dementia disorders in the oldest-old: an autopsy study
TL;DR: This retrospective study using strict morphologic criteria confirmed increased prevalence of AD with age, but mild decline at age 90+, and progressive decline of VD, while AD + vascular pathologies including MIX showed considerable age-related increase, confirming that mixed pathologies account for most dementia cases in very old persons.
Journal ArticleDOI
Clinicopathological analysis of dementia disorders in the elderly
TL;DR: AD/SDAT had its highest incidence in a psychiatric population, MID and PD + SDAT in general and geriatric hospital cohorts, and the results of other recent studies emphasize the need for more appropriate clinical and pathological criteria in the diagnosis of the dementias.
Book ChapterDOI
Neuropathology of Alzheimer's disease: a critical update.
TL;DR: Although the role of plaques and NFT in the pathogenesis of AD remains undetermined, clinicopathological correlative studies have shown that both lesions, if present in sufficient numbers, particularly in the neocortex, are considered the best morphological signposts for AD.
Journal ArticleDOI
The enigma of vascular cognitive disorder and vascular dementia
TL;DR: No validated neuropathologic criteria for VaD are available, and a large variability across laboratories still exists in the procedures for morphologic examination and histology techniques, suggesting different pathogenesis of both types of lesions.