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Kyung Hoon Paik

Researcher at Samsung Medical Center

Publications -  30
Citations -  658

Kyung Hoon Paik is an academic researcher from Samsung Medical Center. The author has contributed to research in topics: Peritoneal dialysis & Ghrelin. The author has an hindex of 12, co-authored 30 publications receiving 612 citations. Previous affiliations of Kyung Hoon Paik include Sungkyunkwan University.

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Journal ArticleDOI

Inflammation and cachexia in chronic kidney disease

TL;DR: Higher serum levels of pro-inflammatory cytokines may be caused by reduced renal function, volume overload, oxidative or carbonyl stress, decreased levels of antioxidants, increased susceptibility to infection in uremia, and the presence of comorbid conditions.
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Identification of mutations in the GNPTA (MGC4170) gene coding for GlcNAc-phosphotransferase alpha/beta subunits in Korean patients with mucolipidosis type II or type IIIA.

TL;DR: Mucolipidosis types II and III are autosomal recessive inherited diseases caused by a deficiency in the lysosomal enzyme N‐acetylglucosamine‐1 phosphotransferase, which adds phosphate to function as a recognition marker for the uptake and transport of lysOSomal enzymes.
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Primary focal segmental glomerular sclerosis in children:clinical course and prognosis

TL;DR: A more prolonged use of corticosteroid therapy and early introduction of cyclosporin A (CsA) may improve the prognosis for primary FSGS in patients with initial steroid nonresponsiveness.
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Correlation between fasting plasma ghrelin levels and age, body mass index (BMI), BMI percentiles, and 24-hour plasma ghrelin profiles in Prader-Willi syndrome.

TL;DR: The data show that there may be a suppressive (or up-regulating) controlling mechanism of ghrelin secretion in obese (or lean) PWS children, and it is hoped that the data may further explain the mechanisms underlying the insatiable appetite and obesity characteristic of PWS.
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Suppression of Acylated Ghrelin during Oral Glucose Tolerance Test Is Correlated with Whole-Body Insulin Sensitivity in Children with Prader-Willi Syndrome

TL;DR: The results suggest that AG is sensitively suppressed by insulin and that this suppression correlated with insulin sensitivity in Prader-Willi syndrome children.