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L.F. Rodarte

Researcher at National Autonomous University of Mexico

Publications -  11
Citations -  435

L.F. Rodarte is an academic researcher from National Autonomous University of Mexico. The author has contributed to research in topics: Taenia solium & Taenia crassiceps. The author has an hindex of 9, co-authored 11 publications receiving 409 citations.

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Cysticercosis: identification and cloning of protective recombinant antigens

TL;DR: The DNA and the deduced amino acid sequence (100 amino acids) of the first protective antigen (KETc7) of potential interest for T. solium pig cysticercosis prevention are reported here.
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Castration and pregnancy of rural pigs significantly increase the prevalence of naturally acquired Taenia solium cysticercosis.

TL;DR: Endocrinological conditions characterized by low levels of androgens or high levels of female hormones probably influence the susceptibility of pigs to T. solium cysticercosis as observed in mice infected with Taenia crassiceps.
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Experimental Taenia solium cysticercosis in pigs: characteristics of the infection and antibody response

TL;DR: The results show that pigs inoculated at 49 and 60 days of age became infected to different degrees and reacted differently to the presence of parasites, suggesting that older animals are more resistant to the infection.
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Immunization of pigs against Taenia solium cysticercosis: factors related to effective protection

TL;DR: In all trials, however, immunized pigs harboured more damaged cysticerci than controls, indicating that immunization does induce some restrictions to parasite these are eventually overwhelmed by other parasite-promoting factors.
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Diagnosis of porcine cysticercosis: a comparative study of serological tests for detection of circulating antibody and viable parasites.

TL;DR: In heavily infected pigs, both antigens and antibodies were detected at least 29 days and up to 200 days post-infection (pi), while in lightly infected pigs antigen and antibody were first observed between 61-97 days pi, while the levels of the serum antigen and antibodies varied with the intensity of the infection.