L
Laura J. Frishman
Researcher at University of Houston
Publications - 127
Citations - 7263
Laura J. Frishman is an academic researcher from University of Houston. The author has contributed to research in topics: Electroretinography & Retina. The author has an hindex of 48, co-authored 122 publications receiving 6693 citations. Previous affiliations of Laura J. Frishman include University of California, San Francisco & Northwestern University.
Papers
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Journal Article
The photopic negative response of the macaque electroretinogram: reduction by experimental glaucoma.
TL;DR: The cornea-negative PhNR of the photopic ERG depends on spiking activity and is reduced in experimental glaucoma when visual sensitivity losses are still mild, but its slow timing raises the possibility that it could be mediated by glia.
Journal Article
Retinal Origins of the Primate Multifocal ERG: Implications for the Human Response
TL;DR: A model of the waveform of the human mfERG is proposed, which suggests that the wave form can be understood as a combination of overlapping ON- and OFF-bipolar cell contributions combined with smaller contributions from inner retina and photoreceptors.
Journal ArticleDOI
Response linearity and kinetics of the cat retina: the bipolar cell component of the dark-adapted electroretinogram.
John G. Robson,Laura J. Frishman +1 more
TL;DR: The time course of the leading edge of the PII response can be interpreted to indicate that the mechanism generating PII introduces three stages of temporal integration in addition to the three stages that are provided by the mechanism of the rod photoreceptors.
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The Scotopic Threshold Response of the Dark‐Adapted Electroretinogram of the Mouse
TL;DR: ERG components of proximal retinal origin that are more sensitive to test flashes and adapting backgrounds than PII provide the ‘threshold’ negative and positive (b‐wave) responses of the mouse dark‐adapted ERG.
Journal Article
The photopic negative response of the flash electroretinogram in primary open angle glaucoma.
TL;DR: PhNR amplitudes in POAG patients are smaller than those of normal subjects, and there is a potential role for the PhNR in early detection and possibly in monitoring the progression of glaucomatous damage.