Laura R. Abad

TL;DR: Osmotin either induced spore lysis, inhibited spore germination or reduced germling viability in seven fungal species that exhibited some degree of sensitivity in hyphal growth inhibition tests, suggesting that the cell wall may be a component of the mechanism by which osmotin permeabilizes the plasma membrane and kills fungal cells.

TL;DR: It is demonstrated that fungal cell wall proteins are determinants of resistance to antifungal PR-5 proteins, indicating that the cell membrane interacts specifically with osmotin and facilitates its action.

TL;DR: The purified recombinant protein exhibited antifungal activity comparable to a class I chitinase purified from NaCl-adapted tobacco cells.

TL;DR: It is shown that transgenic tobacco and potato plants that overexpressed the truncated osmotin protein exhibited resistance to Phytophthora infestans and that in plants overexpressing a C-terminal 20 amino acid truncated Osmotin gene, osmoton was totally secreted into the extracellular matrix.

TL;DR: The results suggest that the signal transduction pathways for elicitation of defense responses by exogenously applied heat-stable nonpathogenic mycelial extract and SA/MeJA overlap at the point of PR protein induction but are not identical.