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Leryn J. Reynolds

Researcher at Old Dominion University

Publications -  25
Citations -  191

Leryn J. Reynolds is an academic researcher from Old Dominion University. The author has contributed to research in topics: Internal medicine & Medicine. The author has an hindex of 7, co-authored 18 publications receiving 151 citations. Previous affiliations of Leryn J. Reynolds include University of Missouri & University of Kentucky.

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Obesity, type 2 diabetes, and impaired insulin-stimulated blood flow: role of skeletal muscle NO synthase and endothelin-1.

TL;DR: Higher basal skeletal muscle expression of ET-1 and reduced peNOS/eNOS may contribute to a reduced insulin-stimulated leg blood flow response in obese T2D patients.
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Developmental Origins of Health Span and Life Span: A Mini-Review

TL;DR: This work has shown that maternal factors, such as environmental toxicant exposure, diet, stress, exercise, age at conception, and longevity have the potential to influence age-associated diseases such as cardiovascular disease, obesity, diabetes, and cancer risk in offspring.
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Acute inactivity impairs glycemic control but not blood flow to glucose ingestion.

TL;DR: Acute reductions in physical activity impaired GC and insulin sensitivity; however, blood flow responses to an OGTT were not affected and a 1-d return to activity was not sufficient to normalize GC after 5 d of reduced daily physical activity.
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A pilot study examining the effects of consuming a high-protein vs normal-protein breakfast on free-living glycemic control in overweight/obese ‘breakfast skipping’ adolescents

TL;DR: The data suggest that the daily addition of a HP breakfast, containing 35 g of high-quality protein, has better efficacy at improving free-living glycemic control compared with a NP breakfast in overweight/obese, but otherwise healthy, ‘breakfast skipping’ adolescents.
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The influence of reduced insulin sensitivity via short-term reductions in physical activity on cardiac baroreflex sensitivity during acute hyperglycemia

TL;DR: It is demonstrated that acute elevations in plasma glucose and insulin can impair spontaneous cardiac BRS in young, healthy subjects, and that reductions in cardiac B RS following acute hyperglycemia are unaffected by reduced insulin sensitivity via short-term reductions in physical activity.