L
Lewis J. Rubin
Researcher at University of California, San Diego
Publications - 373
Citations - 60316
Lewis J. Rubin is an academic researcher from University of California, San Diego. The author has contributed to research in topics: Pulmonary hypertension & Bosentan. The author has an hindex of 101, co-authored 370 publications receiving 57044 citations. Previous affiliations of Lewis J. Rubin include University of Texas Health Science Center at San Antonio & Silver Spring Networks.
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Guías de Práctica Clínica sobre el diagnóstico y tratamiento de la hipertensión arterial pulmonar
Nazzareno Galiè,Adam Torbicki,Robyn J. Barst,Philippe Dartevelle,Sheila G. Haworth,Tim Higenbottam,Horst Olschewski,Andrew J. Peacock,Giuseppe G. Pietra,Lewis J. Rubin,Gérald Simonneau +10 more
TL;DR: Evaluación de la hipertensión arterial pulmonar (tipo, capacidad de ejercicio, hemodinámica) and riesgo y enfermedades asociadas.
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Validation of the 6 min walk test according to the OMERACT filter: a systematic literature review by the EPOSS-OMERACT group
Jérôme Avouac,Otylia Kowal-Bielecka,David Pittrow,Doerte Huscher,Frank Behrens,Christopher P. Denton,Ivan Foeldvari,Marc Humbert,Marco Matucci-Cerinic,Peter Nash,Christian Opitz,Lewis J. Rubin,James R. Seibold,Oliver Distler,Daniel E. Furst +14 more
TL;DR: Current evidence suggests partial validation of the 6MWT in PAH-SSc according to the OMERACT filter, but further dedicated studies are needed to validate completely the 5 min walk test, taking into account the comorbidities interfering with the 6 MWT.
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Clinical worsening in trials of pulmonary arterial hypertension: results and implications.
TL;DR: There is a need for a clear and uniform definition of TTCW that can be tailored to the study population being investigated; the endpoint may require adaptation for patients in different functional classes and with different causes.
Contrasting Mechanisms Contributing to Comparable Alterations with Acetate and Bicarbonate Buffers
TL;DR: It is concluded that transitory hypoventilation contributes to comparable hypoxemia with both acetate and bicarbonate dialysates by different mechanisms, and neither dialysate prevents post-dialysis hypoxemic, which is associated with an increased alveolar-arterial oxygen gradient resulting from a mechanism that remains to be elucidated.
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Gas exchange during dialysis: Contrasting mechanisms contributing to comparable alterations with acetate and bicarbonate buffers
TL;DR: In this article, the short and long-term effects of bicarbonate and acetate dialysate on gas exchange were compared in eight stable patients undergoing dialysis using a closed, proportioning system and a double-blind, crossover study design.