L
Lourens Robbers
Researcher at VU University Medical Center
Publications - 29
Citations - 711
Lourens Robbers is an academic researcher from VU University Medical Center. The author has contributed to research in topics: Myocardial infarction & Medicine. The author has an hindex of 11, co-authored 19 publications receiving 583 citations.
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Journal ArticleDOI
Magnetic resonance imaging-defined areas of microvascular obstruction after acute myocardial infarction represent microvascular destruction and haemorrhage
Lourens Robbers,Elise S. Eerenberg,Paul F. Teunissen,Paul F. Teunissen,Paul F. Teunissen,Matthijs F. Jansen,Matthijs F. Jansen,Matthijs F. Jansen,Maurits R. Hollander,Maurits R. Hollander,Maurits R. Hollander,Anton J.G. Horrevoets,Anton J.G. Horrevoets,Anton J.G. Horrevoets,Paul Knaapen,Paul Knaapen,Paul Knaapen,R. Nijveldt,R. Nijveldt,R. Nijveldt,Martijn W. Heymans,Martijn W. Heymans,Martijn W. Heymans,Marcel Levi,Albert C. van Rossum,Albert C. van Rossum,Albert C. van Rossum,Hans W.M. Niessen,Hans W.M. Niessen,Hans W.M. Niessen,C. Bogdan Marcu,C. Bogdan Marcu,C. Bogdan Marcu,Aernout M. Beek,Aernout M. Beek,Aernout M. Beek,Niels van Royen,Niels van Royen,Niels van Royen +38 more
TL;DR: The contrast-devoid core of revascularized STEMI contains extensive erythrocyte extravasation with microvascular damage andAttenuating the reperfusion-induced haemorrhage may be a novel target in future adjunctive STEMI treatment.
Journal ArticleDOI
A proinflammatory monocyte response is associated with myocardial injury and impaired functional outcome in patients with ST-segment elevation myocardial infarction: monocytes and myocardial infarction.
Anja M. van der Laan,Alexander Hirsch,Lourens Robbers,Robin Nijveldt,Ingrid Lommerse,Ronak Delewi,Pieter A. van der Vleuten,Bart J. Biemond,Jaap Jan Zwaginga,Jaap Jan Zwaginga,Wim J. van der Giessen,Felix Zijlstra,Albert C. van Rossum,Carlijn Voermans,C. Ellen van der Schoot,Jan J. Piek +15 more
TL;DR: An association between a proinflammatory monocyte response, characterized by high levels of classical monocytes, and severe myocardial injury and poor functional outcome after STEMI is shown.
Journal ArticleDOI
Left ventricular thrombus formation after acute myocardial infarction as assessed by cardiovascular magnetic resonance imaging
Ronak Delewi,Robin Nijveldt,Alexander Hirsch,Constantin B. Marcu,Lourens Robbers,Mariëlla E.C.J. Hassell,Rianne H.A. de Bruin,Jim Vleugels,Anja M. van der Laan,Berto J. Bouma,René A. Tio,Jan G.P. Tijssen,Albert C. van Rossum,Felix Zijlstra,Jan J. Piek +14 more
TL;DR: LV thrombus still occurs in a substantial amount of patients after PCI-treated MI, especially in larger infarct sizes, and routine TTE had a low sensitivity for the detection of LV thrombi and the interobserver variation of TTE was large.
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Doppler-Derived Intracoronary Physiology Indices Predict the Occurrence of Microvascular Injury and Microvascular Perfusion Deficits After Angiographically Successful Primary Percutaneous Coronary Intervention
Paul F. Teunissen,Guus A. de Waard,Maurits R. Hollander,Lourens Robbers,Ibrahim Danad,P. Stefan Biesbroek,Raquel P. Amier,Mauro Echavarria-Pinto,Alicia Quirós,Christopher Broyd,Martijn W. Heymans,Robin Nijveldt,Adriaan A. Lammertsma,Pieter G. Raijmakers,Cornelis P. Allaart,Jorrit S. Lemkes,Yolande Appelman,Koen M. Marques,Jean G.F. Bronzwaer,Anton J.G. Horrevoets,Albert C. van Rossum,Javier Escaned,Aernout M. Beek,Paul Knaapen,Niels van Royen +24 more
TL;DR: Doppler-flow–derived physiological indices of coronary resistance (HMR) and extravascular compression (pressure at zero flow) obtained immediately after successful primary PCI predict MVI and decreased PET myocardial blood flow.
Journal ArticleDOI
T1 mapping shows increased extracellular matrix size in the myocardium due to amyloid depositions.
Lourens Robbers,Emma N. Baars,Wessel P. Brouwer,Aernout M. Beek,Mark B.M. Hofman,Hans W.M. Niessen,Albert C. van Rossum,C. Bogdan Marcu +7 more
TL;DR: T1-mapping is a cardiac MR technique, which allows absolute quantification of T1 values of the myocardium and enables assessment of ECM expansion present in cardiac amyloidosis, a systemic infiltrative disorder in which insoluble protein fibrils are deposited in the extracellular matrix.