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M. A. Salam

Researcher at International Centre for Diarrhoeal Disease Research, Bangladesh

Publications -  77
Citations -  1850

M. A. Salam is an academic researcher from International Centre for Diarrhoeal Disease Research, Bangladesh. The author has contributed to research in topics: Population & Vibrio cholerae. The author has an hindex of 19, co-authored 65 publications receiving 1738 citations.

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Large epidemic of cholera-like disease in Bangladesh caused by Vibrio cholerae 0139 synonym Bengal

TL;DR: A large epidemic of cholera-like disease in Bangladesh that is due to a V cholerae non-01 strain seems to have pandemic potential and is described, which suggests that the population has no previous immunological experience of the organism.
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Mortality in severely malnourished children with diarrhoea and use of a standardised management protocol.

TL;DR: Compared with non-protocol management, the standardised protocol resulted in fewer episodes of hypoglycaemia, less need for intravenous fluids, and a 47% reduction in mortality.
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Association of Enterotoxigenic Bacteroides fragilis Infection with Inflammatory Diarrhea

TL;DR: In vitro and in vivo data suggest that enterotoxigenic Bacteroides fragilis (ETBF) may contribute to the burden of colonic inflammatory diarrheal disease, and ETBF infection is a newly recognized cause of inflammatory diarrhea in children and adults.
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Comparison of immune responses in patients infected with Vibrio cholerae O139 and O1.

TL;DR: The results suggest that despite having a polysaccharide capsule, V. cholerae O139 induces systemic and intestine-derived ASC responses in peripheral blood comparable to those seen in patients with V.Cholera O1 disease, which reflects the lack of cross-protection between the infections caused by the two serogroups.
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Comparison of the vibriocidal antibody response in cholera due to Vibrio cholerae O139 Bengal with the response in cholera due to Vibrio cholerae O1.

TL;DR: After modification of the procedure, which involved the use of specific bacterial strains, a lower bacterial inoculum, and increased amounts of complement, the vibriocidal antibody responses to V. cholerae O139 were measured and significant elevations in the homologous antibody response were found only in the convalescent-phase sera from both groups of patients with cholera.