M
M. De Vos
Researcher at Ghent University Hospital
Publications - 187
Citations - 5576
M. De Vos is an academic researcher from Ghent University Hospital. The author has contributed to research in topics: Medicine & Inflammatory bowel disease. The author has an hindex of 42, co-authored 153 publications receiving 5284 citations. Previous affiliations of M. De Vos include Ghent University & University of Leeds.
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Journal ArticleDOI
A positive response to infliximab in Crohn disease: Association with a higher systemic inflammation before treatment but not with-308 TNF gene polymorphism
Edouard Louis,Severine Vermeire,Paul Rutgeerts,M. De Vos,A. Van Gossum,Pierre Pescatore,R. Fiasse,P. Pelckmans,Hendrik Reynaert,G. D'Haens,Michel Malaise,Jacques Belaiche +11 more
TL;DR: A positive clinical response to infliximab was associated with a higher CRP level before treatment in the population of Crohn disease patients, but there was no relevant association with -308 TNF gene polymorphism.
Journal ArticleDOI
Association between polymorphism in IgG Fc receptor IIIa coding gene and biological response to infliximab in Crohn's disease
Edouard Louis,Z. El Ghoul,Severine Vermeire,Sebastien Dall'ozzo,Paul Rutgeerts,Gilles Paintaud,Jacques Belaiche,M. De Vos,A. Van Gossum,Jean-Frederic Colombel,Hervé Watier +10 more
TL;DR: To test the hypothesis of an association between polymorphism in FCGR3A (the gene coding for FcγRIIIa, which is expressed on macrophages and natural killer cells, is involved in antibody‐dependent cell‐mediated cytotoxicity) and response to infliximab in Crohn's disease.
Journal ArticleDOI
Inflammatory Bowel Disease A Positive Response to Infliximab in Crohn Disease: Association with a Higher Systemic Inflammation Before Treatment But Not With -308 TNF Gene Polymorphism
Edouard Louis,Severine Vermeire,Paul Rutgeerts,M. De Vos,A. Van Gossum,Paul Pescatore,R. Fiasse,P. Pelckmans,H. Reynaert,G. D'Haens,Michel Malaise,Jacques Belaiche +11 more
TL;DR: A positive clinical response to infliximab was associated with a higher CRP level before treatment in the population of Crohn disease patients, but there was no relevant association with-308 TNF gene polymorphism.
Journal ArticleDOI
Long-term evolution of gut inflammation in patients with spondyloarthropathy
TL;DR: The evolution of the intestinal inflammation observed in patients with SpA supports the etiopathogenetic role of the gut in SpA and has a beneficial effect on articular activity by controlling gut inflammation, but it cannot prevent evolution to overt IBD.
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Ileocolonoscopic Findings in Seronegative Spondylarthropathies
TL;DR: Some of the patients with seronegative spondylarthropathies suffer from a subclinical form of Crohn's disease of which the joint symptoms are the unique clinical manifestation, support for this hypothesis was provided by the results of a repeat ileocolonoscopy, disclosing a strong association between the presence of gut inflammation on biopsy and the persistence of joint inflammation.