M
M. Ian Gilmour
Researcher at United States Environmental Protection Agency
Publications - 113
Citations - 4458
M. Ian Gilmour is an academic researcher from United States Environmental Protection Agency. The author has contributed to research in topics: Lung injury & Immune system. The author has an hindex of 36, co-authored 103 publications receiving 3974 citations. Previous affiliations of M. Ian Gilmour include University of North Carolina at Chapel Hill & Research Triangle Park.
Papers
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Journal ArticleDOI
How exposure to environmental tobacco smoke, outdoor air pollutants, and increased pollen burdens influences the incidence of asthma.
TL;DR: It is illustrated how environmental tobacco smoke, outdoor air pollution, and climate change may act as environmental risk factors for the development of asthma and mechanistic explanations for how some of these effects can occur are provided.
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Oxidant Generation and Lung Injury after Particulate Air Pollutant Exposure Increase with the Concentrations of Associated Metals
Robert J. Pritchard,Andrew J. Ghio,James R. Lehmann,Darrell W. Winsett,Jeffrey S. Tepper,Patricia Park,M. Ian Gilmour,Kevin L. Dreher,Daniel L. Costa +8 more
TL;DR: In this article, the hypothesis that particulate air pollutants are associated with metals that have a capacity to transport electrons and that biologic activity of the particulates can correlate with the concentrations of these metals was tested.
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Prenatal air pollution exposure induces neuroinflammation and predisposes offspring to weight gain in adulthood in a sex-specific manner
Jessica L. Bolton,Susan H. Smith,Nicole C. Huff,M. Ian Gilmour,W. Michael Foster,Richard L. Auten,Staci D. Bilbo +6 more
TL;DR: Prenatal air pollution exposure can “program” offspring for increased susceptibility to diet‐induced weight gain and neuroinflammation in adulthood in a sex‐specific manner, as tested in mice exposed to filtered air or diesel exhaust on embryonic days 9‐17.
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Metal composition of ambient PM2.5 influences severity of allergic airways disease in mice.
TL;DR: Metal composition of ambient PM(2.5) influences the severity of allergic respiratory disease and is consistent with epidemiologic findings and shown to be influential on lung injury parameters and proinflammatory cytokines.
Journal ArticleDOI
Ozone Exposure Increases Circulating Stress Hormones and Lipid Metabolites in Humans.
Desinia B. Miller,Andrew J. Ghio,Edward D. Karoly,Lauren N. Bell,Samantha J. Snow,Michael C. Madden,Joleen M. Soukup,Wayne E. Cascio,M. Ian Gilmour,Urmila P. Kodavanti +9 more
TL;DR: As in rodents, acute ozone exposure increased stress hormones and globally altered peripheral lipid metabolism in humans, likely through activation of a neurohormonally mediated stress response pathway.