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M

M. L.H. Katayama

Researcher at University of São Paulo

Publications -  11
Citations -  118

M. L.H. Katayama is an academic researcher from University of São Paulo. The author has contributed to research in topics: Cellular differentiation & HL60. The author has an hindex of 5, co-authored 11 publications receiving 107 citations.

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Human Semaphorin 6B [(HSA)SEMA6B], A Novel Human Class 6 Semaphorin Gene: Alternative Splicing and All-Trans-Retinoic Acid-Dependent Downregulation in Glioblastoma Cell Lines

TL;DR: It is observed that (HSA)SEMA6B is highly expressed in human brain and at lower levels in a variety of other tissues and upon prolonged treatment with all-trans-retinoic acid, an anti-tumor and differentiation-inducing agent.
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Gene trio signatures as molecular markers to predict response to doxorubicin cyclophosphamide neoadjuvant chemotherapy in breast cancer patients.

TL;DR: The combined expression of MTSS1, RPL37 and SMYD2, as evaluated by real-time RT-PCR, is a potential candidate to predict response to neoadjuvant doxorubicin and cyclophosphamide in breast cancer patients.
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Expression of E-cadherin, Snail and Hakai in epithelial cells isolated from the primary tumor and from peritumoral tissue of invasive ductal breast carcinomas.

TL;DR: This is the first report simultaneously analyzing CDH1, SNAI1 and HAKAI mRNA expression in matched tumor and peritumoral samples from patients with IDC, and no clear pattern of their expression could distinguish the invasive tumor compartment from its adjacent normal tissue.
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Vitamin D3 binding activity during leukemic cell differentiation.

TL;DR: The data suggest that 1,25(OH)2D3 receptor levels could be considered a marker of functional immaturity, in these cells.
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Differential regulation of vitamin D receptor expression in distinct leukemic cell lines upon phorbol ester-induced growth arrest

TL;DR: The data suggest that VDR expression cannot be explained simply as a reflection of the leukemic cell growth state, since VDR levels were down-regulated only in HL-60 cells.