M
M. P. Stern
Researcher at University of Texas Health Science Center at San Antonio
Publications - 82
Citations - 7041
M. P. Stern is an academic researcher from University of Texas Health Science Center at San Antonio. The author has contributed to research in topics: Population & Diabetes mellitus. The author has an hindex of 42, co-authored 82 publications receiving 6932 citations. Previous affiliations of M. P. Stern include Texas Biomedical Research Institute.
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Hyperinsulinaemia: the key feature of a cardiovascular and metabolic syndrome
TL;DR: It is concluded that insulin sensitivity, glucose tolerance, blood pressure, body fat mass and distribution, and serum lipids are a network of mutually interrelated functions and an insulin resistance syndrome underlies each and all of the six disorders carrying an increased risk of coronary artery disease.
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A prospective analysis of the HOMA model : The Mexico City diabetes study
TL;DR: It is concluded that the HOMA provides a useful model to assess ²-cell function in epidemiological studies and that it is important to take into account the degree of IR in assessing insulin secretion.
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Birthweight and adult health outcomes in a biethnic population in the USA
TL;DR: Low birthweight could be a major independent risk factor for the development of adult chronic conditions commonly associated with insulin resistance in the general population.
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Hyperinsulinemia in a Population at High Risk for Non-Insulin-Dependent Diabetes Mellitus
TL;DR: The data suggest that, like other populations at high risk for NIDDM such as Pima Indians and Micronesians, Mexican Americans have more hyperinsulinemia than can be accounted for by their adiposity.
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Microalbuminuria. Potential marker for increased cardiovascular risk factors in nondiabetic subjects
S. M. Haffner,M. P. Stern,M K Gruber,Helen P. Hazuda,Braxton D. Mitchell,Judith K. Patterson +5 more
TL;DR: The level of microalbuminuria was determined in 316 nondiabetic subjects from the San Antonio Heart Study, a population-based study of diabetes and cardiovascular risk factors, suggesting that an increased atherogenic risk factor pattern exists even in normotensive subjects with micro Albuminuria.