M
M. T. Rivera
Researcher at Université libre de Bruxelles
Publications - 26
Citations - 740
M. T. Rivera is an academic researcher from Université libre de Bruxelles. The author has contributed to research in topics: Trypanosoma cruzi & Parasitemia. The author has an hindex of 15, co-authored 22 publications receiving 708 citations. Previous affiliations of M. T. Rivera include Pasteur Institute & Oswaldo Cruz Foundation.
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Journal Article
Endogenous IFN-gamma is required for resistance to acute Trypanosoma cruzi infection in mice.
TL;DR: This study clearly indicates that IFN-gamma is produced early in acute T. cruzi infection and exerts a protective effect that is probably independent from the humoral immune response.
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Trypanosoma cruzi infection in mice induces a polyisotypic hypergammaglobulinaemia and parasite-specific response involving high IgG2a concentrations and highly avid IgG1 antibodies.
TL;DR: The results indicate that T. cruzi infection in mice induces a polyisotypic humoral immune response, dominated by some antibody isotypes, with major differences in concentrations and functional avidities, which could be of crucial importance in determining the outcome of infection.
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Progressive Chagas' cardiomyopathy is associated with low selenium levels.
M. T. Rivera,Andrea Pereira De Souza,Alejandro Hasslocher Moreno,Sérgio Salles Xavier,Juliana A. S. Gomes,Manoel Otávio da Costa Rocha,Rodrigo Correa-Oliveira,Jean Neve,Jean Vanderpas,Tania C. Araújo-Jorge +9 more
TL;DR: The decrease in Se in chagasic patients seems to be a biological marker for Trypanosoma cruzi infection and related to the progression of pathology.
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Trace elements, innate immune response and parasites.
M. T. Rivera,Andrea Pereira De Souza,Tania C. Araújo-Jorge,Solange L. de Castro,Jean Vanderpas +4 more
TL;DR: Some interactions of trace elements with innate immunity and acute inflammatory response are reviewed in this article with a special focus on selenium deficiency and Trypanosoma cruzi infection.
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Modulation induced by estradiol in the acute phase of Trypanosoma cruzi infection in mice.
TL;DR: Estradiol interferes with T. cruzi infection, and females infected during an estradiol-descending phase (meta-oestrus) survived longer than those infected during other phases of the oestrous cycle.