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Ma Easter Joy Sajo

Researcher at Yonsei University

Publications -  14
Citations -  204

Ma Easter Joy Sajo is an academic researcher from Yonsei University. The author has contributed to research in topics: Oxidative stress & Hairless. The author has an hindex of 7, co-authored 14 publications receiving 140 citations. Previous affiliations of Ma Easter Joy Sajo include University of the Philippines Baguio & University of the Philippines.

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Immunotoxicity of silicon dioxide nanoparticles with different sizes and electrostatic charge

TL;DR: Interestingly, the small-sized and negatively charged SiO2 NPs showed the most potent in vivo immunotoxicity by way of suppressing the proliferation of lymphocytes, depressing the killing activity of NK cells, and decreasing proinflammatory cytokine production, thus leading to immunosuppression.
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Hydrogen Water Drinking Exerts Antifatigue Effects in Chronic Forced Swimming Mice via Antioxidative and Anti-Inflammatory Activities.

TL;DR: This study shows antifatigue effects of HW drinking in chronic forced swimming mice via metabolic coordination and immune-redox balance and drinking HW could be applied to the alternative and safety fluid remedy for chronic fatigue control.
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Antioxidant and Anti-Inflammatory Effects of Shungite against Ultraviolet B Irradiation-Induced Skin Damage in Hairless Mice.

TL;DR: Results clearly show that shungite has an antioxidant and anti-inflammatory action against UVB-induced skin damage in hairless mice.
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The balneotherapy effect of hydrogen reduced water on UVB-mediated skin injury in hairless mice

TL;DR: The bathing with HRW significantly reduced the levels of skin damage, as well as increased activity of glutathione peroxidase and the effect of HRW on cytokine network in the skin after UVB exposure revealed thatHRW significantly decreased the level of inflammatory cytokines such as IL-1β, IL-6, TNF-α and IFN-γ.
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Positive Effects of hydrogen water on 2,4-dinitrochlorobenzene-induced atopic dermatitis in NC/Nga mice.

TL;DR: The collective results indicate that HW suppresses DNCB-induced AD in NC/Nga mice via redox balance and immune modulation and could be a safe clinical fluid treatment for AD.