Manoel Sarno

TL;DR: This case report provides evidence that in addition to microcephaly, there may be a link between Zika virus infection and hydrops fetalis and fetal demise and systematic investigation of spontaneous abortions and stillbirths is warranted.

TL;DR: The data corroborate ZIKV-associated congenital disease and an association of low SES and ZikV infection and suggest that population immunity caused cessation of the outbreak.

TL;DR: A systematic review and meta‐analysis on the relationship between excess weight and risk of recurrent pregnancy loss and the common immunological mechanisms of these two conditions revealed that obese women with a history of RPL have a high risk of future pregnancy losses.

TL;DR: Insight is gained into the scope and severity of microcephaly due to presumed congenital Zika syndrome (CZS), as well as challenges in confirming this association, which supports the assertion that Zika virus causes a distinct congenital syndrome.

TL;DR: It is suggested that multitypic DENV infection may protect from, rather than enhance, development of CZS, and historical DENV genomic data refuted the hypothesis that unique genome signatures for northeastern Brazil explain the uneven dispersion of CzS cases.