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Maria Eugenia Oliva

Researcher at National University of the Littoral

Publications -  20
Citations -  443

Maria Eugenia Oliva is an academic researcher from National University of the Littoral. The author has contributed to research in topics: Insulin resistance & Insulin. The author has an hindex of 9, co-authored 20 publications receiving 365 citations.

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Dietary chia seed ( Salvia hispanica L.) rich in α-linolenic acid improves adiposity and normalises hypertriacylglycerolaemia and insulin resistance in dyslipaemic rats

TL;DR: Dietary chia seed prevented the onset of dyslipidaemia and IR in the rats fed the SRD for 3 weeks – glycaemia did not change and dyslipIDAemia andIR in the long-term SRD-fed rats were normalised without changes in insulinaemia when chia Seed provided the dietary fat during the last 2 months of the feeding period.
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Dietary Salba (Salvia hispanica L) seed rich in α-linolenic acid improves adipose tissue dysfunction and the altered skeletal muscle glucose and lipid metabolism in dyslipidemic insulin-resistant rats.

TL;DR: Chia seed reversed the impaired insulin stimulated glycogen synthase activity, glycogen, glucose-6-phosphate and GLUT-4 protein levels as well as insulin resistance and dyslipidemia.
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Dietary chia seed induced changes in hepatic transcription factors and their target lipogenic and oxidative enzyme activities in dyslipidaemic insulin-resistant rats.

TL;DR: New data is provided regarding some key mechanisms related to the fate of hepatic fatty acid metabolism that seem to be involved in the effect of dietary chia seed in preventing and normalising/improving dyslipidaemia and liver steatosis in an insulin-resistant rat model.
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Mechanisms Involved in the Improvement of Lipotoxicity and Impaired Lipid Metabolism by Dietary α-Linolenic Acid Rich Salvia hispanica L (Salba) Seed in the Heart of Dyslipemic Insulin-Resistant Rats

TL;DR: Normalization of dyslipidemia and IR by chia reduces plasma fatty acids (FAs) availability, suggesting that a different milieu prevents the robust translocation of FAT/CD36.
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Sucrose‐rich feeding during rat pregnancy‐lactation and/or after weaning alters glucose and lipid metabolism in adult offspring

TL;DR: The hypothesis that early life exposure to SRD is associated with changes in lipid and glucose metabolism, leading to an unfavourable profile in adulthood, is supported, regardless of whether offspring consumed an SRD after weaning.